Dysregulated hematopoietic stem and progenitor cell activity promotes interleukin-23-driven chronic intestinal inflammation.
In interleukin-23 (IL-23)-dependent colitis, there is excessive accumulation of short-lived neutrophils and inflammatory monocytes in the intestine. It is unknown whether this reflects changes in mature cell populations or whether the IL-23-driven colitogenic T cell program regulates upstream hemato...
Κύριοι συγγραφείς: | Griseri, T, McKenzie, B, Schiering, C, Powrie, F |
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Μορφή: | Conference item |
Έκδοση: |
2012
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Παρόμοια τεκμήρια
Παρόμοια τεκμήρια
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Dysregulated Hematopoietic Stem and Progenitor Cell Activity Promotes Interleukin-23-Driven Chronic Intestinal Inflammation
ανά: Griseri, T, κ.ά.
Έκδοση: (2012) -
Exacerbated hematopoietic stem and progenitor cell activity during intestinal inflammation
ανά: Griseri, T, κ.ά.
Έκδοση: (2011) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells
ανά: Schiering, C, κ.ά.
Έκδοση: (2010) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells
ανά: Ahern, P, κ.ά.
Έκδοση: (2011) -
Interleukin-23 drives intestinal inflammation through direct activity on T cells.
ανά: Ahern, P, κ.ά.
Έκδοση: (2010)