Sclerostin regulation, microarchitecture, and advanced glycation end-products in the bone of elderly women with type 2 diabetes

Sclerostin regulation, microarchitecture, and advanced glycation end-products in the bone of elderly women with type 2 diabetes

Increased circulating sclerostin and accumulation of advanced glycation end‐products (AGEs) are two potential mechanisms underlying low bone turnover and increased fracture risk in type 2 diabetes (T2D). Whether the expression of the sclerostin‐encoding SOST gene is altered in T2D, and whether it is...

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Bibliographic Details
Main Authors: Piccoli, A, Cannata, F, Strollo, R, Pedone, C, Leanza, G, Russo, F, Greto, V, Isgrò, C, Quattrocchi, CC, Massaroni, C, Silvestri, S, Vadalà, G, Bisogno, T, Denaro, V, Pozzilli, P, Tang, SJ, Silva, MJ, Conte, C, Papalia, R, Maccarrone, M, Napoli, N
Format: Journal article
Language:English
Published: Wiley 2020

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