Global improvements in both central and peripheral vascular function after short-term folic acid treatment in patients with coronary artery disease - A cardiovascular MR study

Bibliographic Details
Main Authors:	Shirodaria, C, Lee, J, Antoniades, C, Jackson, C, Robson, M, Francis, J, Pillai, R, Ratnatunga, C, Channon, K
Format:	Conference item
Published:	2005

Short-term folic acid treatment improves central and peripheral vascular function in patients with coronary artery disease - A cardiovascular magnetic resonance study
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Published: (2006)

No additional improvement in vascular function with high dose vs. low dose folic acid in coronary artery disease - Implications for HOPE-2
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Improvement in vascular function with low-dose folic acid supplementation: Implications for folate therapy and dietary fortification
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Global improvement of vascular function and redox state with low-dose folic acid: implications for folate therapy in patients with coronary artery disease.
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Folic acid induces maximal vascular effects at dosage equivalent to the recommended daily allowance (RDA): Implications for folic acid treatment in fortified populations
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Direct improvement of endothelial function and superoxide production in human vessels, by oral folic acid treatment: A double blind placebo controlled study
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Chronic treatment with folic acid improves endothelial function and decreases superoxide production in human vessels. A double blind placebo control study
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Plasma biopterins are independent predictors of arterial distensibility and endothelial function in coronary artery disease - A cardiovascular MR study
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Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
by: Antoniades, C, et al.
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Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
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Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
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Vascular MR imaging reveals early improvements in aortic and carotid function following initiation of statin therapy in patients with coronary artery disease
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Rapid effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
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5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
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Direct effects of 5-methyl-tetrahydrofolate on vascular endotheliurn and superoxide production in human arteries and veins
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Multimodal cardiovascular magnetic resonance quantifies regional variation in vascular structure and function in patients with coronary artery disease: relationships with coronary disease severity.
by: Kylintireas, I, et al.
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The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
by: Antoniades, C, et al.
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Systemic and vascular oxidation limits the efficacy of oral tetrahydrobiopterin treatment in patients with coronary artery disease.
by: Cunnington, C, et al.
Published: (2012)

Systemic and vascular oxidation limits the efficacy of oral tetrahydrobiopterin treatment in patients with coronary artery disease
by: Cunnington, C, et al.
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Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
by: Antoniades, C, et al.
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Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
by: Antoniades, C, et al.
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Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
by: Antoniades, C, et al.
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Relationships between vascular/plasma biopterins and endothelial nitric oxide synthase coupling, endothelial function and inflammation, in human atherosclerosis
by: Antoniades, C, et al.
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Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
by: Antoniades, C, et al.
Published: (2008)

Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
by: Antoniades, C, et al.
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Statin treatment rapidly reduces aortic atherosclerosis and improves vascular function: A cardiovascular magnetic resonance study
by: Lee, J, et al.
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Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
by: Antoniades, C, et al.
Published: (2006)

Multi-modal magnetic resonance imaging quantifies atherosclerosis and vascular dysfunction in patients with type 2 diabetes mellitus.
by: Lee, J, et al.
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Chronic oral tetrahydrobiopterin therapy in coronary artery disease elevates total biopterin levels but does not improve redox status or vascular function: a randomized placebo-controlled trial
by: Cunnington, C, et al.
Published: (2010)

5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
by: Antoniades, C, et al.
Published: (2006)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
by: Antoniades, C, et al.
Published: (2006)

Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway
by: Antoniades, C, et al.
Published: (2008)

5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
by: Antoniades, C, et al.
Published: (2006)

CHRONIC ORAL TETRAHYDROBIOPTERIN TREATMENT IN PATIENTS WITH CORONARY ARTERY DISEASE ELEVATES TOTAL BIOPTERIN LEVELS BUT DOES NOT IMPROVE BIOPTERIN REDOX STATUS OR VASCULAR FUNCTION: A RANDOMISED PLACEBO-CONTROLLED TRIAL
by: Cunnington, C, et al.
Published: (2010)

Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
by: Antoniades, C, et al.
Published: (2009)

MTHFR 677 C>T Polymorphism reveals functional importance for 5-methyltetrahydrofolate, not homocysteine, in regulation of vascular redox state and endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
by: Antoniades, C, et al.
Published: (2009)

Altered plasma versus vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function, and inflammation.
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Serum adiponectin regulates vascular O2-generation and NO bioavailability in patients with coronary atherosclerosis
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Global improvements in both central and peripheral vascular function after short-term folic acid treatment in patients with coronary artery disease - A cardiovascular MR study

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