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Analysis of Lyn/CD22 double-de...
Čujuhandieđut
Deakstadieđáhus
Sádde šleađgaboasttain
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Doalvvo čujuhusa
Doalvun: RefWorks
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Doalvun: EndNote
Bissovaš liŋka
Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22-independent pathways affecting BCR regulation and B cell survival (vol 35, pg 3655, 2005)
Bibliográfalaš dieđut
Váldodahkkit:
Ferry, H
,
Crockford, T
,
Silver, K
,
Rust, N
,
Goodnow, C
,
Cornall, R
Materiálatiipa:
Journal article
Almmustuhtton:
2005
Oažžasuvvandieđut
Govvádus
Geahča maid
Bargiidšearbma
Govvádus
Čoahkkáigeassu:
Geahča maid
Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22-independent pathways affecting BCR regulation and B cell survival.
Dahkki: Ferry, H, et al.
Almmustuhtton: (2005)
Erratum: Analysis of Lyn/CD22 double-deficient B cells in vivo demonstrates Lyn- and CD22- independent pathways affecting BCR regulation and B cell survival (European Journal of Immunology (2005) vol. 35 (12) (3714) 10.1002/eji.200535247)
Dahkki: Ferry, H, et al.
Almmustuhtton: (2005)
Regulation of B cell antigen receptor signaling by the Lyn/CD22/SHP1 pathway.
Dahkki: Cornall, R, et al.
Almmustuhtton: (1999)
Polygenic autoimmune traits: Lyn, CD22, and SHP-1 are limiting elements of a biochemical pathway regulating BCR signaling and selection.
Dahkki: Cornall, R, et al.
Almmustuhtton: (1998)
Spontaneous class switching and B cell hyperactivity increase autoimmunity against intracellular self antigen in Lyn-deficient mice.
Dahkki: Silver, K, et al.
Almmustuhtton: (2006)