Does the K-ATP channel participate in the regulation of glucagon secretion?

Does the K-ATP channel participate in the regulation of glucagon secretion?

Bibliografski detalji
Glavni autori:	Bokvist, K, Olsen, H, Hoy, M, Gotfredsen, C, Buschard, K, Rorsman, P, Gromada, J
Format:	Journal article
Izdano:	1999

Slični predmeti

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Gi2 proteins couple somatostatin receptors to low-conductance K+ channels in rat pancreatic alpha-cells.
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ATP-sensitive K+ channel-dependent regulation of glucagon release and electrical activity by glucose in wild-type and SUR1-/- mouse alpha-cells.
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K(ATP)-channels and glucose-regulated glucagon secretion.
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Imidazoline NNC77-0074 stimulates insulin secretion and inhibits glucagon release by control of Ca(2+)-dependent exocytosis in pancreatic alpha- and beta-cells.
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Inhibition of ATP-regulated K(+)-channels by a photoactivatable ATP-analogue in mouse pancreatic beta-cells.
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Evidence from in situ human alpha-cells supporting intrinsic K-ATP channel-dependent regulation of glucagon secretion
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GLUCOSE REGULATION OF GLUCAGON-SECRETION INVOLVES ACTIVATION OF GABAA-RECEPTOR CL--CHANNELS
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Nateglinide, but not repaglinide, stimulates growth hormone secretion from rat pituitary cells.
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Somatostatin but not insulin is an important paracrine regulator of glucagon secretion in rat A-cells
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Glucose-inhibition of glucagon secretion involves activation of GABAA-receptor chloride channels.
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DEFECTIVE REGULATION OF GLUCOSE-REGULATED AND ATP-REGULATED K-CHANNELS RESULTS IN DISTURBED GLUCOSE SENSITIVITY IN FETAL B-CELLS
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Separate processes mediate nucleotide-induced inhibition and stimulation of the ATP-regulated K(+)-channels in mouse pancreatic beta-cells.
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GLUCOSE DOES NOT BLOCK ATP-REGULATED K+-CHANNEL ACTIVITY IN FETAL B-CELLS
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Glucagon stimulates exocytosis in mouse and rat pancreatic alpha-cells by binding to glucagon receptors.
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Adrenaline stimulates secretion in rat pancreatic alpha-cells by PKA-dependent acceleration of Ca2+ influx through 1-type Ca2+- channels
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