Cumulative negative effects of donor brain death and subsequent ischemia/reperfusion on kidney graft injury and function.

Cumulative negative effects of donor brain death and subsequent ischemia/reperfusion on kidney graft injury and function.

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Bibliografiska uppgifter
Huvudupphovsmän:	Nijboer, W, Schuurs, T, Swart, B, Ottens, P, Leuvenink, H, Ploeg, R
Materialtyp:	Conference item
Publicerad:	2007

Liknande verk

Cumulative negative effects of donor brain death and subsequent ischemia/reperfusion on kidney graft injury and function
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Effect of brain death on gene expression and tissue activation in human donor kidneys.
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Donor pretreatment with carbamylated erythropoietin in a brain death model reduces inflammation more effectively than erythropoietin while preserving renal function.
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Brain death induces apoptosis in donor liver of the rat.
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Gradual Rewarming with Gradual Increase in Pressure during Machine Perfusion after Cold Static Preservation Reduces Kidney Ischemia Reperfusion Injury.
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Dysfunction of Blood-Brain Barrier in Deceased Brain Dead Donors Leads to Increased Serum IL-6 and a Decreased Outcome after Transplantation.
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Local renal complement C3 induction by donor brain death is associated with reduced renal allograft function after transplantation.
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Crosstalk between Complement and Toll-like Receptor Activation in Relation to Donor Brain Death and Renal Ischemia-Reperfusion Injury (vol 11, pg 660, 2011)
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Correction: Gradual Rewarming with Gradual Increase in Pressure during Machine Perfusion after Cold Static Preservation Reduces Kidney Ischemia Reperfusion Injury.
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Brain death induces inflammation in the donor intestine.
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Early events in kidney donation: progression of endothelial activation, oxidative stress and tubular injury after brain death.
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The Effect of Hypothermic Machine Perfusion to Ameliorate Ischemia-Reperfusion Injury in Donor Organs
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Static cold storage preservation of ischemically damaged kidneys a comparison between IGL-1 and UW solution in a rat DCD donormodel
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ARA290, a non-erythropoietic EPO derivative, attenuates renal ischemia/reperfusion injury
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In Situ Hypothermic Oxygenated Machine Perfusion with UW in Deceased Cardiac Death Donors Improves Early Renal Function
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Local renal upregulation of complement C3 and fibrinogen as part of the acute phase response in brain-dead donors
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