Eosinophils are major effector cells for the development of transplant arteriosclerosis and depend on the presence of IL-4.

Eosinophils are major effector cells for the development of transplant arteriosclerosis and depend on the presence of IL-4.

Bibliografski detalji
Glavni autori:	Ensminger, S, Witzke, O, Spriewald, B, Sorensen, H, Morris, P, Rahemtulla, A, Rose, M, Wood, K
Format:	Journal article
Izdano:	2000

Slični predmeti

Critical role for IL-4 in the development of transplant arteriosclerosis in the absence of CD40-CD154 costimulation.
od: Ensminger, S, i dr.
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Indirect allorecognition plays a pivotal role in the pathogenesis of transplant arteriosclerosis.
od: Ensminger, S, i dr.
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Indirect allorecognition can play an important role in the development of transplant arteriosclerosis.
od: Ensminger, S, i dr.
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CD8+ T cells contribute to the development of transplant arteriosclerosis despite CD154 blockade.
od: Ensminger, S, i dr.
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Increased expression of transforming growth factor-beta and eosinophil infiltration is associated with the development of transplant arteriosclerosis in long-term surviving cardiac allografts.
od: Spriewald, B, i dr.
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Kinetics of transplant arteriosclerosis in MHC-Class I mismatched and fully allogeneic mouse aortic allografts.
od: Ensminger, S, i dr.
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The persistence of transplant arteriosclerosis despite CD154 blockade.
od: Billing, J, i dr.
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Mouse endothelial CD40 expression does not play a role during the development of transplant arteriosclerosis.
od: Ensminger, S, i dr.
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Intragraft interleukin-4 mRNA expression after short-term CD154 blockade may trigger delayed development of transplant arteriosclerosis in the absence of CD8+ T cells.
od: Ensminger, S, i dr.
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Neutralizing interleukin-4 prevents transplant arteriosclerosis mediated by indirect pathway T cells under CD40-CD154 costimulation blockade.
od: Spriewald, B, i dr.
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Development of a combined cardiac and aortic transplant model to investigate the development of transplant arteriosclerosis in the mouse.
od: Ensminger, S, i dr.
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Platelet-endothelial cell adhesion molecule-1 (CD31) expression on donor endothelial cells attenuates the development of transplant arteriosclerosis.
od: Ensminger, S, i dr.
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Increased transplant arteriosclerosis in the absence of CCR7 is associated with reduced expression of Foxp3.
od: Ensminger, S, i dr.
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Comparison of the effects of exposure to a single or multiple donor alloantigens in the development of transplant arteriosclerosis.
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Investigation into the onset and progression of transplant arteriosclerosis in a mice aortic retransplantation model.
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Regulation of transplant arteriosclerosis by CD25+CD4+ T cells generated to alloantigen in vivo.
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Complex arterial reconstruction for pancreas transplantation in recipients with advanced arteriosclerosis.
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Ly-6C(hi) monocytes: a potential target for preventing transplant arteriosclerosis?
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In vivo prevention of transplant arteriosclerosis by ex vivo-expanded human regulatory T cells.
od: Nadig, SN, i dr.
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CD4+ regulatory T cells generated in vitro with IFN-{gamma} and allogeneic APC inhibit transplant arteriosclerosis.
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IL-5 antagonism reverses priming and activation of eosinophils in severe eosinophilic asthma
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Low-dose rapamycin treatment increases the ability of human regulatory T cells to inhibit transplant arteriosclerosis in vivo.
od: Hester, J, i dr.
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Low-dose rapamycin treatment increases the ability of human regulatory T cells to inhibit transplant arteriosclerosis in vivo.
od: Hester, J, i dr.
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Ex vivo expanded human treg inhibit the development of transplant arteriosclerosis in a humanized in vivo model
od: Nadig, SN, i dr.
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Inflammatory Ly-6Chi monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients
od: Schiopu, A, i dr.
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Inflammatory Ly-6C(hi) monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients.
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Eosinophil migration is enhanced towards IL-5 and eotaxin in COPD
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Long-term allograft survival following intrathymic application of plasmid encoding endoplasmic reticulum signal sequence deleted MHC class I alloantigen
od: Spriewald, B, i dr.
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Intrathymic delivery of plasmid-encoding endoplasmic reticulum signal-sequence-deleted MHC class I alloantigen can induce long-term allograft survival.
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Sputum and bronchial submucosal IL-13 expression in asthma and eosinophilic bronchitis.
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Neutrophils recruited by IL-22 in peripheral tissues function as TRAIL-dependent antiviral effectors against MCMV
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Neutrophils recruited by IL-22 in peripheral tissues function as TRAIL-dependent antiviral effectors against MCMV.
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Linked unresponsiveness: early cytokine gene expression profiles in cardiac allografts following pretreatment of recipients with bone marrow cells expressing donor MHC alloantigen.
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IL-6 effector function of group 2 innate lymphoid cells (ILC2) is NOD2 dependent
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IL-4-induced eosinophil accumulation in rat skin is dependent on endogenous TNF-alpha and alpha 4 integrin/VCAM-1 adhesion pathways.
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Induction of transplantation tolerance converts potential effector T cells into graft-protective regulatory T cells.
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Deletion of the major GATA1 enhancer HS 1 does not affect eosinophil GATA1 expression and eosinophil differentiation.
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High viral burden in the presence of major HIV-specific CD8(+) T cell expansions: evidence for impaired CTL effector function.
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Suppression mediated by anergic CD4+ T cells requires stimulation by MHC-peptide complexes and can be induced in the presence of costimulation.
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Seasonality of eosinophilic and non-eosinophilic exacerbations of COPD
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