Elevating high-density lipoprotein cholesterol in apolipoprotein E-deficient mice remodels advanced atherosclerotic lesions by decreasing macrophage and increasing smooth muscle cell content.

Míreanna comhchosúla

• Apolipoprotein AI overexpression remodels large, complex atherosclerotic lesions to have characteristics of more stable plaques in apoE-deficient mice
  de réir: Rong, J, et al.
  Foilsithe / Cruthaithe: (2001)
• High-density lipoproteins retard the progression of atherosclerosis and favorably remodel lesions without suppressing indices of inflammation or oxidation.
  de réir: Choudhury, R, et al.
  Foilsithe / Cruthaithe: (2004)
• Laser capture microdissection analysis of gene expression in macrophages from atherosclerotic lesions of apolipoprotein E-deficient mice.
  de réir: Trogan, E, et al.
  Foilsithe / Cruthaithe: (2002)
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  de réir: Choudhury, R, et al.
  Foilsithe / Cruthaithe: (2002)
• Human apolipoprotein A-I transgene expression retards the progression of atherosclerosis in apolipoprotein E deficient mice fed Western-type diet (WD)
  de réir: Choudhury, R, et al.
  Foilsithe / Cruthaithe: (2001)