Manipulating creatine levels post-myocardial infarction--chronic effects on left ventricular remodeling.

Manipulating creatine levels post-myocardial infarction--chronic effects on left ventricular remodeling.

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Neubauer, S, Horn, M, Schnackerz, K
التنسيق:	Journal article
اللغة:	English
منشور في:	1998

مواد مشابهة

Chronic high-dose creatine feeding does not attenuate left ventricular remodeling in rat hearts post-myocardial infarction.
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Creatine kinase knockout mice show left ventricular hypertrophy and dilatation, but unaltered remodeling post-myocardial infarction.
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Left-ventricular hypertrophy and post-infarct remodelling in mice lacking creatine kinase isoenzyme assessed by magnetic resonance imaging
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Preservation of left ventricular mechanical function and energy metabolism in rats after myocardial infarction by the angiotensin-converting enzyme inhibitor quinapril.
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Left ventricular remodeling in patients with right ventricular myocardial infarction
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Left ventricular remodeling in patients with right ventricular myocardial infarction
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Functional and energetic consequences of chronic myocardial creatine depletion by beta-guanidinopropionate in perfused hearts and in intact rats.
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Changes of creatine kinase gene expression in rat heart post-myocardial infarction.
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Mitochondrial creatine kinase is critically necessary to maintain a normal profile of myocardial high-energy phosphates
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Plasma Circulating Extracellular RNAs in Left Ventricular Remodeling Post-Myocardial Infarction
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In-Hospital Levels of Circulating MicroRNAs as Potential Predictors of Left Ventricular Remodeling Post-Myocardial Infarction
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Effects of chronic dietary creatine feeding on cardiac energy metabolism and on creatine content in heart, skeletal muscle, brain, liver and kidney.
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Serial cine-magnetic resonance imaging of left ventricular remodeling after myocardial infarction in rats.
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Living without creatine: unchanged exercise capacity and response to chronic myocardial infarction in creatine-deficient mice.
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nNOS gene deletion exacerbates pathological left ventricular remodelling and functional deterioration after myocardial infarction
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nNOS gene deletion exacerbates pathological left ventricular remodeling and functional deterioration after myocardial infarction.
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Left ventricular lipomatous metaplasia following myocardial infarction.
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Post Myocardial Infarction Left Ventricular Giant Pseudoaneurysm
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Ischaemic preconditioning prevents left ventricular remodelling in patients with acute myocardial infarction
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Is longitudinal strain associated with left ventricular remodeling in patients with acute myocardial infarction?
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Impact of myocardial hemorrhage on left ventricular function and remodeling in patients with reperfused acute myocardial infarction
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Regulation of Myocardial Creatine Uptake by Intra- and Extracellular Creatine Levels
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Chronic phosphocreatine depletion by the creatine analogue beta-guanidinopropionate is associated with increased mortality and loss of ATP in rats after myocardial infarction.
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Beneficial effect of ischemic preconditioning on post-infarction left ventricular remodeling and global left ventricular function
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