Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway

Bibliographische Detailangaben
Hauptverfasser:	Antoniades, C, Shirodaria, C, Leeson, P, Baarholm, O, Antonopoulos, A, Pillai, R, Ratnatunga, C, Tousoulis, D, Refsum, H, Stefanadis, C, Channon, K
Format:	Conference item
Veröffentlicht:	2008

Plasma levels of sCD40-ligand interact with homocysteine biosynthetic pathway and predict eNOS coupling in human vessels: effects on vascular redox state
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Plasma asymmetrical-dimethyl-arginine affects eNOS vascular superoxide radical production and nitric oxide bioavailability in human arteries and veins
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Genetic Polymorphism On Methyl-Tetrahydrofolate-Reductase Affects Vascular Superoxide Generation and Nitric Oxide Bioavailability By Regulating eNOS Coupling in Human Arteries and Veins
von: Antoniades, C, et al.
Veröffentlicht: (2009)

The common genetic polymorphism C677T of methyltetrahydrofolate reductase regulates nitric oxide bioavailability and vascular superoxide production via modulation of eNOS coupling in human vessels
von: Antoniades, C, et al.
Veröffentlicht: (2007)

Genetic variability of MTHFR affects vascular redox and nitric oxide bioavailability in human vessels, by modifying eNOS coupling
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Vascular Homocysteine Versus 5-Methyl-Tetrahydrofolate As Regulators of Vascular Redox and Endothelial Function in Human Vessels
von: Antoniades, C, et al.
Veröffentlicht: (2009)

Plasma asymmetrical-dimethyl-arginine (ADMA) regulates nitric oxide bioavailability and vascular superoxide production in human vessels
von: Antoniades, C, et al.
Veröffentlicht: (2007)

Vascular Homocysteine vs 5-Methyl-Tetrahydrofolate as Regulators of Vascular Redox and Endothelial Function in Human Vessels
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Genetic Variability of Methyl-Tetrahydrofolate-Reductase Modifies eNOS Coupling in Human Arteries and Veins: Effects on Vascular Superoxide Generation and Nitric Oxide Bioavailabllity
von: Antoniades, C, et al.
Veröffentlicht: (2008)

MTHFR 677 C>T Polymorphism reveals functional importance for 5-methyltetrahydrofolate, not homocysteine, in regulation of vascular redox state and endothelial function in human atherosclerosis.
von: Antoniades, C, et al.
Veröffentlicht: (2009)

5-Methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production by improving eNOS coupling and increasing tetrahydrobiopterin bioavailability in human vessels in vivo
von: Antoniades, C, et al.
Veröffentlicht: (2006)

5-methyl-tetrahydrofolic acid increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling in human vessels in vivo: Effects on intracellular tetrahydrobiopterin oxidation
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Low- and high- dose treatment with folic acid, equally improve endothelial function and decrease superoxide production in human vessels via tetrahydrobiopterin-mediated eNOS coupling
von: Antoniades, C, et al.
Veröffentlicht: (2007)

5-methyl-tetrahydrofolate increases nitric oxide bioavailability and decreases superoxide production by improving eNOS coupling and preventing the intracellular oxidation of tetrahy-drobiopterin, in human vessels in vivo
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Chronic treatment with folic acid improves nitric oxide bioavailability and decreases superoxide production in human vessels, through tetrahydrobiopterin-mediated improvement of eNOS coupling
von: Antoniades, C, et al.
Veröffentlicht: (2007)

Polymorphism of MTHFR gene affects vascular redox and nitric oxide bioavailability by modifying eNOS coupling
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Plasma asymmetrical-dimethyl-arginine (ADMA) as a regulator of vascular redox and nitric oxide bioavailability in human vessels
von: Antoniades, C, et al.
Veröffentlicht: (2007)

5-methyl-tetrahydrofolate increases eNOS activity by preventing peroxynitrite-induced oxidation of tetrahydrobiopterin in human vessels
von: Antoniades, C, et al.
Veröffentlicht: (2006)

5-methyl-tetrahydrofolate acid improves endothelial function and decreases superoxide production in vessels from patients with coronary artery disease
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Interrelationships between vascular superoxide and adiponectin biosynthesis in perivascular adipose tissue in human atherosclerosis
von: Bakogiannis, C, et al.
Veröffentlicht: (2010)

Association of plasma asymmetrical dimethylarginine (ADMA) with elevated vascular superoxide production and endothelial nitric oxide synthase uncoupling: implications for endothelial function in human atherosclerosis.
von: Antoniades, C, et al.
Veröffentlicht: (2009)

Intravenous infusion of 5-methyl-tetrahydrofolate rapidly improves eNOS Coupling. In human vessels by increasing tetrahydrobiopterin bioavailability
von: Antoniades, C, et al.
Veröffentlicht: (2006)

5-Methyl-tetrahydrofolic acid rapidly improves endothelial function and superoxide production in human atherosclerosis: Effects on tetrahyd robiopterin-dependent eNOS coupling
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Direct improvement of endothelial function and superoxide production in human vessels, by oral folic acid treatment: A double blind placebo controlled study
von: Antoniades, C, et al.
Veröffentlicht: (2005)

5-methyltetrahydrofolate rapidly improves endothelial function and decreases superoxide production in human vessels: effects on vascular tetrahydrobiopterin availability and endothelial nitric oxide synthase coupling.
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Homocysteine lowering: any use in atherosclerosis?
von: Antoniades, C, et al.
Veröffentlicht: (2007)

A Novel Haplotype on GCH1 Gene, Encoding GTP Cyclohydrolase 1, Regulates Vascular Biopterin Synthesis, eNOS Coupling and Vascular Redox in Human Vessels
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Chronic treatment with folic acid improves endothelial function and decreases superoxide production in human vessels. A double blind placebo control study
von: Shirodaria, C, et al.
Veröffentlicht: (2005)

Direct effects of 5-methyl-tetrahydrofolate on vascular endotheliurn and superoxide production in human arteries and veins
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Oral Atorvastatin Treatment Improves NO Bioavailability and Vascular Redox State in the Human Arterial Wall, via a Tetrahydrobiopterin-Mediated Improvement of eNOS Coupling
von: Antoniades, C, et al.
Veröffentlicht: (2011)

Homocysteine and coronary atherosclerosis: from folate fortification to the recent clinical trials.
von: Antoniades, C, et al.
Veröffentlicht: (2009)

Rapid effects of 5-methyl-tetrahydrofolic acid on endothelial function and vascular superoxide production in human coronary artery disease
von: Antoniades, C, et al.
Veröffentlicht: (2006)

Relationships between vascular/plasma biopterins and endothelial nitric oxide synthase coupling, endothelial function and inflammation, in human atherosclerosis
von: Antoniades, C, et al.
Veröffentlicht: (2008)

Is vascular superoxide triggering adiponectin biosynthesis in perivascular adipose tissue in patients with atherosclerosis?
von: Bakogiannis, C, et al.
Veröffentlicht: (2010)

Folic acid induces maximal vascular effects at dosage equivalent to the recommended daily allowance (RDA): Implications for folic acid treatment in fortified populations
von: Antoniades, C, et al.
Veröffentlicht: (2007)

Altered plasma vs. vascular biopterins in human atherosclerosis reveal relationships between endothelial nitric oxide synthase coupling, endothelial function and inflammation
von: Antomades, C, et al.
Veröffentlicht: (2007)

RAPID, DIRECT EFFECTS OF STATIN TREATMENT ON ARTERIAL REDOX STATE AND NITRIC OXIDE BIOAVAILABILITY IN HUMAN ATHEROSCLEROSIS VIA TETRAHYDROBIOPTERIN-MEDIATED ENOS COUPLING
von: Antoniades, C, et al.
Veröffentlicht: (2011)

Rapid Effects of Atorvastatin on Tetrahydrobiopterin-Mediated eNOS Coupling in Human Arteries, is Mediated by Stimulation of Vascular GTP-Cyclohydrolase
von: Antoniades, C, et al.
Veröffentlicht: (2011)

Genetic variation in GCH1, encoding GTP-cyclohydrolase 1, regulates biopterin levels and endothelial function in atherosclerosis
von: Van, A, et al.
Veröffentlicht: (2008)

Soluble CD40-ligand levels are associated with vascular superoxide production and eNOS coupling in human vessels: Interrelationships with homocysteine biosynthetic pathway

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