The ClC-5 knockout mouse model of Dent's disease has renal hypercalciuria and increased bone turnover.

Lignende værker

• Mice lacking renal chloride channel, CLC-5, are a model for Dent's disease, a nephrolithiasis disorder associated with defective receptor-mediated endocytosis.
  af: Wang, S, et al.
  Udgivet: (2000)
• STIM1fl/fl Ksp-Cre Mouse has Impaired Renal Water Balance
  af: Liudmila Cebotaru, et al.
  Udgivet: (2016-06-01)
• Intra-renal and subcellular distribution of the human chloride channel, CLC-5, reveals a pathophysiological basis for Dent's disease.
  af: Devuyst, O, et al.
  Udgivet: (1999)
• Comparative ontogeny, processing, and segmental distribution of the renal chloride channel, ClC-5.
  af: Jouret, F, et al.
  Udgivet: (2004)
• Mutational analysis of CLC-5, cofilin and CLC-4 in patients with Dent's disease.
  af: Wu, F, et al.
  Udgivet: (2009)