CA2+ MOVEMENTS DURING STIMULATION OF INSULIN RELEASE BY REMOVAL OF EXTRACELLULAR NA+

CA2+ MOVEMENTS DURING STIMULATION OF INSULIN RELEASE BY REMOVAL OF EXTRACELLULAR NA+

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Rorsman, P, Andersson, T, Berggren, P, Hellman, B
Μορφή:	Journal article
Έκδοση:	1980

Παρόμοια τεκμήρια

Ca2+ transport in pancreatic beta-cells during glucose stimulation of insulin secretion.
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Calcium and pancreatic beta-cell function. XI. Modification of 45Ca fluxes by Na+ removal.
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CLONIDINE INHIBITION OF GLUCOSE-STIMULATED INSULIN-RELEASE INVOLVES REDUCTION OF THE CA-2+ CURRENT
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Ca(2+)-induced Ca2+ release in insulin-secreting cells.
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Reduction of the cytosolic calcium activity in clonal insulin-releasing cells exposed to glucose.
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Inhibition of glucose-stimulated insulin release by alpha 2-adrenoceptor activation is parallelled by both a repolarization and a reduction in cytoplasmic free Ca2+ concentration.
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MEMBRANE-POTENTIAL, TRANSMEMBRANE CA-45 FLUXES, CYTOPLASMIC FREE CA-2+ CONCENTRATION ([CA-2+]I) AND INSULIN RELEASE BY TRANSPLANTABLE RAT INSULINOMA CELLS
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Suppression of insulin release by galanin and somatostatin is mediated by a G-protein. An effect involving repolarization and reduction in cytoplasmic free Ca2+ concentration.
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Extracellular ATP increases cytoplasmic free Ca2+ concentration in clonal insulin-producing RINm5F cells. A mechanism involving direct interaction with both release and refilling of the inositol 1,4,5-trisphosphate-sensitive Ca2+ pool.
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Manganese accumulation in pancreatic beta-cells and its stimulation by glucose.
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Measurements of membrane potential, transmembrane 45Ca fluxes, cytoplasmic free Ca2+ concentration and insulin release by transplantable rat insulinoma cells maintained in tissue culture.
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MEASUREMENTS OF CYTOPLASMIC PH IN INSULIN-RELEASING CELLS WITH INTRACELLULARLY TRAPPED PHENOL RED
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Glucose stimulates the entry of Ca2+ into the insulin-producing beta cells but not into the glucagon-producing alpha 2 cells.
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Direct measurements of increased free cytoplasmic Ca2+ in mouse pancreatic beta-cells following stimulation by hypoglycemic sulfonylureas.
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CYCLIC-AMP POTENTIATION OF INSULIN RELEASE FROM PANCREATIC BETA-CELLS IS NOT MEDIATED BY AN INCREASED CYTOSOLIC CA-2+ ACTIVITY
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Stimulation of insulin release by repaglinide and glibenclamide involves both common and distinct processes.
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Insulin prevents counter-regulatory glucagon secretion by stimulation of somatostatin release
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SERUM FROM INSULIN-DEPENDENT DIABETICS INCREASE L-TYPE CA-2+-CHANNEL ACTIVITY AND CYTOPLASMIC FREE CA-2+ IN INSULIN-PRODUCING CELLS
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Cyclic AMP potentiates glucose-induced insulin release from mouse pancreatic islets without increasing cytosolic free Ca2+.
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Imidazoline NNC77-0074 stimulates insulin secretion and inhibits glucagon release by control of Ca(2+)-dependent exocytosis in pancreatic alpha- and beta-cells.
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Insulin secretion: a high-affinity Ca2+ sensor after all?
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