Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide

Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide

מידע ביבליוגרפי
Main Authors:	Hijazo, R, Hale, AB, Liu, X, Suffredini, S, Bendall, J, Lim, G, Alp, N, Channon, K, Casadei, B
פורמט:	Conference item
יצא לאור:	2012

פריטים דומים

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Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
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201 Regulation of INOS Function, Redox State and NRF2 Activation by Macrophage GCH1 and Tetrahydrobiopterin.
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
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Hydrogen peroxide differentially modulates cardiac myocyte nitric oxide synthesis
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Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
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