Role of platelet endothelial adhesion molecule (PECAM)-1 in sensing and mediating high temporal gradients of shear stress in arterioles

Role of platelet endothelial adhesion molecule (PECAM)-1 in sensing and mediating high temporal gradients of shear stress in arterioles

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Koller, A, Frangos, J, Kaley, G, Bagi, Z
Formáid:	Conference item
Foilsithe / Cruthaithe:	2006

Míreanna comhchosúla

PECAM-1 mediates NO-dependent dilation of arterioles to high temporal gradients of shear stress.
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Reduced sensitivity wall shear stress in arterioles of PECAM knockout mice due to impaired mechanotransduction
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PPAR gamma activation restores NO-Mediated dilations of coronary Arterioles in type 2 diabetes Mellitus
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Superoxide-NO interaction decreases flow- and agonist-induced dilations of coronary arterioles in Type 2 diabetes mellitus.
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PPARgamma activation, by reducing oxidative stress, increases NO bioavailability in coronary arterioles of mice with Type 2 diabetes.
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Differential effect of Platelet Endothelial Cell Adhesion Molecule-1 (PECAM-1) on leukocyte infiltration during contact hypersensitivity responses
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Low fluid shear stress stimulates the uptake of noxious endothelial extracellular vesicles via MCAM and PECAM‐1 cell adhesion molecules
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Simultaneously increased TxA(2) activity in isolated arterioles and platelets of rats with hyperhomocysteinemia.
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Aldose reductase inhibition reduces endothelial dysfunction and oxidative stress in skeletal muscle arterioles exposed to hyperglycemia
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Increased PGH(2)/TXA(2) synthesis in isolated arterioles and platelets of rats with hyperhomocysteinemia (HHCY).
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Binding of Plasmodium falciparum-infected erythrocytes to soluble platelet endothelial cell adhesion molecule-1 (PECAM-1/CD31): frequent recognition by clinical isolates.
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Serum levels of platelet endothelial cell adhesion molecule-1 (PECAM-1) and soluble vascular endothelial growth factor receptor (sVEGFR)-1 and -2 in HIV associated preeclampsia
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Hydrogen peroxide elicits COX-2-dependent production of constrictor prostaglandins in arterioles of type 2 diabetic mice
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Cloning and Stable Expression of cDNA Coding For Platelet Endothelial Cell Adhesion Molecule -1 (PECAM-1, CD31) in NIH-3T3 Cell Line
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Nitric oxide and hydrogen peroxide contribute to the development of reactive dilation in isolated coronary arterioles
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Tetrahydrobiopterin (BH4) is essential for NO-mediated flow-dependent dilation of isolated skeletal muscle arterioles
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Nitric oxide and H2O2 contribute to reactive dilation of isolated coronary arterioles.
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Platelet PECAM-1 inhibits thrombus formation in vivo.
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Multiple effects of diabetes mellitus on the vasomotor responses of human coronary arterioles
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Intrinsic vascular mechanisms stimulated by changes in pressure and now elicit reactive dilatation of isolated arterioles
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PIEZO1 and PECAM1 interact at cell-cell junctions and partner in endothelial force sensing
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Heparan sulfate proteoglycan glypican-1 and PECAM-1 cooperate in shear-induced endothelial nitric oxide production
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High intraluminal pressure reduces tachyphylaxis to angiotensin II in isolated arterioles
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Conducted vasodilatation in human coronary arterioles
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Impaired flow-induced dilation in skeletal muscle arterioles of rats with hyperhomocysteinemia (HHCY). Role of nitric oxide and constrictor prostanoids.
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Adaptation of vasomotor function of human coronary arterioles to the simultaneous presence of obesity and hypertension.
de réir: Fulop, T, et al.
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PPAR gamma activation improves wall shear stress dependent regulation of coronary arteriolar diameter in type 2 diabetes mellitus
de réir: Bagi, Z, et al.
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Asymmetric dimethylarginine (ADMA) increases the myogenic tone of arterioles due to enhanced release of superoxide
de réir: Toth, J, et al.
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Flow-induced constriction in arterioles of hyperhomocysteinemic rats is due to impaired nitric oxide and enhanced thromboxane A(2) mediation.
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Linking hyperpolarization to endothelial cell calcium events in arterioles.
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Adhesion Molecules ICAM-1 and PECAM-1 as Potential Biomarkers of Central Nervous System Damage in Women Breast Cancer Survivors
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Increased cyclooxygenase-2 expression and prostaglandin-mediated dilation in coronary arterioles of patients with diabetes mellitus.
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Lack of flow-mediated dilation and enhanced angiotensin II-induced constriction in skeletal muscle arterioles of lupus-prone autoimmune mice.
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THE EXPRESSION OF THE ADHESION MOLECULES PECAM, ICAM-1, ICAM-2, ICAM-3, E-SELECTIN AND P-SELECTIN IN BREAST-CANCER ENDOTHELIUM
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Platelet Endothelial Cell Adhesion Molecule-1 Polymorphism in Patients with Bronchial Asthma
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Increased activity of tissue ACE impairs dilation of coronary arterioles in obesity
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Chronic renal failure leads to reduced flow-dependent dilation in isolated rat skeletal muscle arterioles due to lack of NO mediation.
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Collaborative enhancement of antibody binding to distinct PECAM-1 epitopes modulates endothelial targeting.
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Platelet dysfunction in platelet-type von Willebrand disease due to the constitutive triggering of the Lyn-PECAM1 inhibitory pathway
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Targeting platelet inhibition receptors for novel therapies: PECAM-1 and G6b-B
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