Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.

Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.

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Atrial fibrillation (AF) is a growing public health burden, and its treatment remains a challenge. AF leads to electrical remodeling of the atria, which in turn promotes AF maintenance and resistance to treatment. Although remodeling has long been a therapeutic target in AF, its causes remain poorly...

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Bibliographic Details
Main Authors: Reilly, S, Liu, X, Carnicer, R, Recalde, A, Muszkiewicz, A, Jayaram, R, Carena, M, Wijesurendra, R, Stefanini, M, Surdo, N, Lomas, O, Ratnatunga, C, Sayeed, R, Krasopoulos, G, Rajakumar, T, Bueno-Orovio, A, Verheule, S, Fulga, T, Rodriguez, B, Schotten, U, Casadei, B
Format: Journal article
Language:English
Published: American Association for the Advancement of Science 2016

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