Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.

Atrial fibrillation (AF) is a growing public health burden, and its treatment remains a challenge. AF leads to electrical remodeling of the atria, which in turn promotes AF maintenance and resistance to treatment. Although remodeling has long been a therapeutic target in AF, its causes remain poorly...

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Main Authors:	Reilly, S, Liu, X, Carnicer, R, Recalde, A, Muszkiewicz, A, Jayaram, R, Carena, M, Wijesurendra, R, Stefanini, M, Surdo, N, Lomas, O, Ratnatunga, C, Sayeed, R, Krasopoulos, G, Rajakumar, T, Bueno-Orovio, A, Verheule, S, Fulga, T, Rodriguez, B, Schotten, U, Casadei, B
Format:	Journal article
Language:	English
Published:	American Association for the Advancement of Science 2016

Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase by Reilly, S, Liu, X, Carnicer, R, Recalde, A, Muszkiewicz, A, Jayaram, R, Carena, M, Wijesurendra, R, Stefanini, M, Surdo, N, Lomas, O, Ratnatunga, C, Sayeed, R, Krasopoulos, G, Rajakumar, T, Bueno-Orovio, A, Verheule, S, Fulga, T, Rodriguez, B, Schotten, U, Casadei, B

Published 2016

Journal article

Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.

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