Impaired NO-mediation of flow dependent arteriolar dilation in diabetes due to reduced level of tetrahydrobiopterin

Samankaltaisia teoksia

• Transient elevation of glucose concentration reduces NO-mediation of flow-induced arteriolar dilation due to interaction between superoxide and tetrahydrobiopterin (BH4)
  Tekijä: Bagi, Z, et al.
  Julkaistu: (2004)
• Lack of nitric oxide mediation of flow-dependent arteriolar dilation in type I diabetes is restored by sepiapterin.
  Tekijä: Bagi, Z, et al.
  Julkaistu: (2003)
• Tetrahydrobiopterin (BH4) is essential for NO-mediated flow-dependent dilation of isolated skeletal muscle arterioles
  Tekijä: Bagi, Z, et al.
  Julkaistu: (2003)
• Upregulation of Arginase I Reduces Nitric Oxide-mediated Coronary Arteriolar Dilation in Patients With Type 2 Diabetes
  Tekijä: Bagi, Z, et al.
  Julkaistu: (2009)
• Enhanced superoxide production reduces NO-mediation of flow-dependent dilation of isolated coronary arteries in type 2 diabetes mellitus
  Tekijä: Bagi, Z, et al.
  Julkaistu: (2003)