Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1

Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1

Xehetasun bibliografikoak
Egile Nagusiak:	Ashley, E, Bryant, S, Sears, C, Kim, Y, Watkins, H, Casadei, B
Formatua:	Journal article
Argitaratua:	2001

Antzeko izenburuak

Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
nork: Ashley, E, et al.
Argitaratua: (2002)

Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
nork: Zhang, Y, et al.
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
nork: Ashley, E, et al.
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
nork: Ashley, E, et al.
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Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
nork: Sears, C, et al.
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
nork: Sears, C, et al.
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
nork: Sears, C, et al.
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nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
nork: Zhang, Y, et al.
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NEURONAL NITRIC OXIDE SYNTHASE-MEDIATED REGULATION OF MYOCARDIAL REDOX STATE AND beta 3 ADRENERGIC RECEPTOR RESPONSES
nork: Idigo, W, et al.
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Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
nork: Sears, C, et al.
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Insulin resistance in mice lacking neuronal nitric oxide synthase is related to an alpha-adrenergic mechanism
nork: P Turini, et al.
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Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation
nork: Kohlhaas, M, et al.
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
nork: Bryant, S, et al.
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
nork: Sears, C, et al.
Argitaratua: (2000)

Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
nork: Bryant, S, et al.
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Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling.
nork: Sears, C, et al.
Argitaratua: (2003)

Myocardial NOS1 controls the lusitropic response to beta-adrenergic stimulation in vivo and in vitro
nork: Sears, C, et al.
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A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function
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Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression
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Superoxide production by left ventricular myocytes in control and hypertrophied hearts
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Mechanical and electrophysiological properties of murine hypertrophied left ventricular myocytes
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Nitric oxide synthases in heart failure.
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Mechanical and electrophysiological properties of left ventricular myocytes in catecholamine-induced hypertrophy in mice
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Development of Genetically Engineered Mice Lacking All Three Nitric Oxide Synthases
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Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide
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Exercise protects against chronic β-adrenergic remodeling of the heart by activation of endothelial nitric oxide synthase.
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Disruption of inhibitory G-proteins mediates a reduction in atrial beta-adrenergic signaling by enhancing eNOS expression.
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The emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.
nork: Casadei, B
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Nitric-oxide-mediated regulation of cardiac contractility and stretch responses.
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Neuronal nitric oxide synthase and human vascular regulation.
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NO from neuronal NO synthase increases after beta-adrenergic stimulation but does not control mitochondrial respiration
nork: Bergem, S, et al.
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Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.
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Spontaneous pulmonary emphysema in mice lacking all three nitric oxide synthase isoforms
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Involvement of beta3-adrenergic receptors in relaxation mediated by nitric oxide in chicken basilar artery
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Are myocardial eNOS and nNOS involved in the beta-adrenergic and muscarinic regulation of inotropy? A systematic investigation.
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Neonatal Mice Lacking Neuronal Nitric Oxide Synthase Are Less Vulnerable to Hypoxic–Ischemic Injury
nork: Donna M. Ferriero, et al.
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Neuronal nitric oxide synthase and affective disorders
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A critical role for neuronal nitric oxide synthase in the Physiologic response to exercise
nork: De Bono, J, et al.
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BACTERIAL NITRIC OXIDE SYNTHASE
nork: Б. М. Галкін, et al.
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Significance of nitric oxide synthases: Lessons from triple nitric oxide synthases null mice
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