PPAR alpha, free fatty acids and mitochondrial UCP3 in mouse heart

PPAR alpha, free fatty acids and mitochondrial UCP3 in mouse heart

Bibliografski detalji
Glavni autori:	Murray, A, Panagia, M, Hauton, D, Gibbons, G, Clarke, K
Format:	Conference item
Izdano:	2004

Slični predmeti

Plasma free fatty acids and peroxisome proliferator-activated receptor alpha in the control of myocardial uncoupling protein levels.
od: Murray, A, i dr.
Izdano: (2005)

PPAR-alpha activation required for decreased glucose uptake and increased susceptibility to injury during ischemia.
od: Panagia, M, i dr.
Izdano: (2005)

Peroxisome proliferator activated receptor alpha mediates the increased susceptibility to ischemic injury in the diabetic mouse heart
od: Panagia, M, i dr.
Izdano: (2003)

Metabolomics of the interaction between PPAR-alpha and age in the PPAR-alpha-null mouse.
od: Atherton, H, i dr.
Izdano: (2009)

Control of cardiac uncoupling proteins: PPARa-dependence?
od: Murray, A, i dr.
Izdano: (2005)

The effect of omega3 fatty acid supplementation on PPARγ and UCP2 expressions, resting energy expenditure, and appetite in athletes
od: Sara Moradi, i dr.
Izdano: (2021-05-01)

High-fat diet alters physical and mental performance via changes in mitochondrial UCPS
od: Knight, N, i dr.
Izdano: (2008)

PPAR alpha is involved in triiodothyronine-induced cardiac hypertrophy and high-energy phosphate metabolism: A P-31 MRS study of the PPAR alpha-null mouse
od: Zhang, W, i dr.
Izdano: (2006)

Plasma free fatty acids and energy depletion in failing human hearts
od: Murray, A, i dr.
Izdano: (2004)

PPARα suppresses insulin secretion and induces UCP2 in insulinoma cells
od: Karen Tordjman, i dr.
Izdano: (2002-06-01)

Abnormal function and glucose metabolism in the type-2 diabetic db/db mouse heart.
od: Panagia, M, i dr.
Izdano: (2007)

PPAR alpha is Essential for Cardiac Metabolic Adaptation to Chronic Hypoxia
od: Cole, M, i dr.
Izdano: (2010)

Utilisation of triacylglycerol and non-esterified fatty acid by the working rat heart: myocardial lipid substrate preference.
od: Hauton, D, i dr.
Izdano: (2001)

Free fatty acids reduce efficiency in the hypertrophied rat heart
od: Cole, M, i dr.
Izdano: (2005)

Expressions of mRNA and encoded proteins of mitochondrial uncoupling protein genes (UCP1, UCP2, and UCP3) in epicardial and mediastinal adipose tissue and associations with coronary artery disease
od: Claudia Huesca-Gómez, i dr.
Izdano: (2023-01-01)

PPARδ is a fatty acid sensor that enhances mitochondrial oxidation in insulin-secreting cells and protects against fatty acid-induced dysfunction
od: Kim Ravnskjaer, i dr.
Izdano: (2010-06-01)

UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis
od: Jong-Seok Moon, i dr.
Izdano: (2023-03-01)

Carbon monoxide induced PPARγ SUMOylation and UCP2 block inflammatory gene expression in macrophages.
od: Arvand Haschemi, i dr.
Izdano: (2011-01-01)

UCP1 deficiency increases adipose tissue monounsaturated fatty acid synthesis and trafficking to the liver
od: Laura M. Bond, i dr.
Izdano: (2018-02-01)

Mitochondrial Respiration-Dependent ANT2-UCP2 Interaction
od: Tomas A. Schiffer, i dr.
Izdano: (2022-05-01)

Regulation of fatty acid oxidation in mouse cumulus-oocyte complexes during maturation and modulation by PPAR agonists.
od: Kylie R Dunning, i dr.
Izdano: (2014-01-01)

Brown adipocytes promote epithelial mesenchymal transition of neuroblastoma cells by inducing PPAR-γ/UCP2 expression
od: Zhijuan Ge, i dr.
Izdano: (2022-12-01)

PPARγ Inhibits VSMC Proliferation and Migration via Attenuating Oxidative Stress through Upregulating UCP2.
od: Yi Zhou, i dr.
Izdano: (2016-01-01)

Utilisation of fatty acid and triacylglycerol by rat macrophages: the effect of endotoxin.
od: Hauton, D, i dr.
Izdano: (2002)

Metabolomics of the interaction between PPAR‐α and age in the PPAR‐α‐null mouse
od: Helen J Atherton, i dr.
Izdano: (2009-04-01)

A high fat diet increases mitochondrial fatty acid oxidation and uncoupling to decrease efficiency in rat heart.
od: Cole, M, i dr.
Izdano: (2011)

A high fat diet increases mitochondrial fatty acid oxidation and uncoupling to decrease efficiency in rat heart
od: Cole, M, i dr.
Izdano: (2011)

Cardiomyocyte-Restricted Deletion of PPARβ/δ in PPARα-Null Mice Causes Impaired Mitochondrial Biogenesis and Defense, but No Further Depression of Myocardial Fatty Acid Oxidation
od: Jian Liu, i dr.
Izdano: (2011-01-01)

UCP-2 and UCP-3 proteins are differentially regulated in pancreatic beta-cells.
od: Yunfeng Li, i dr.
Izdano: (2008-01-01)

Chronic Fatty Acid Depletion Induces Uncoupling Protein 1 (UCP1) Expression to Coordinate Mitochondrial Inducible Proton Leak in a Human-Brown-Adipocyte Model
od: Yukimasa Takeda, i dr.
Izdano: (2022-06-01)

Trans fatty acids and coronary heart disease.
od: Clarke, R, i dr.
Izdano: (2006)

Correction: UCP-2 and UCP-3 Proteins Are Differentially Regulated in Pancreatic Beta-Cells.
od: Yunfeng Li, i dr.
Izdano: (2022-01-01)

Mitochondrial fission is associated with UCP1 activity in human brite/beige adipocytes
od: Didier F. Pisani, i dr.
Izdano: (2018-01-01)

Upregulation of UCP2 by adiponectin: the involvement of mitochondrial superoxide and hnRNP K.
od: Mingyan Zhou, i dr.
Izdano: (2012-01-01)

PERM1 regulates genes involved in fatty acid metabolism in the heart by interacting with PPARα and PGC-1α
od: Chun-yang Huang, i dr.
Izdano: (2022-08-01)

Astragaloside IV alleviates heart failure via activating PPARα to switch glycolysis to fatty acid β-oxidation
od: Zhiwei Dong, i dr.
Izdano: (2017-06-01)

Fatty acids regulate perilipin5 in muscle by activating PPARδ[S]
od: Christian Bindesb⊘ll, i dr.
Izdano: (2013-07-01)

Fatty acid and triacylglycerol utilisation by perfused rat spleen: differential metabolic fate and the effect of endotoxin.
od: Hauton, D, i dr.
Izdano: (2002)

Mitochondrial Uncoupling Proteins (UCP1-UCP3) and Adenine Nucleotide Translocase (ANT1) Enhance the Protonophoric Action of 2,4-Dinitrophenol in Mitochondria and Planar Bilayer Membranes
od: Kristina Žuna, i dr.
Izdano: (2021-08-01)

TTA is a pan PPAR ligand which markedly stimulates fatty acid oxidation following in vivo administration
od: Khalid, A, i dr.
Izdano: (2008)