Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering

Atorvastatin rapidly reduces myocardial superoxide and peroxynitrite generation in human atherosclerosis, by suppressing NADPH-oxidase activity independently of LDL lowering

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Antoniades, C, Demosthenous, M, Reilly, S, Margaritis, M, Jayaram, R, Nahar, K, Tousoulis, D, Stefanadis, C, Channon, K, Casadei, B
Μορφή:	Journal article
Έκδοση:	2011

Παρόμοια τεκμήρια

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Short-term preoperative treatment with atorvastatin improves myocardial redox state in patients undergoing elective CABG by affecting NADPH oxidase activity and NOS coupling
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Systemic Lipid Peroxydation and Myocardial Superoxide/Peroxynitrite Anions as Predictors of Post-Operative Outcome in Cardiac Surgery
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Preoperative atorvastatin treatment rapidly improves vein graft redox state by inhibiting rac1 and NADPH oxidase activity in patients undergoing elective CABG
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Atorvastatin directly improves vascular redox state by affecting eNOS coupling and NADPH-oxidase activity in human vein grafts ex-vivo
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Perivascular adipose tissue supresses superoxide production in internal mammary artery grafts by regulating Rac1-mediated activation of vascular NADPH-oxidase
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Preoperative atorvastatin treatment in CABG patients rapidly improves vein graft redox state by inhibition of Rac1 and NADPH-oxidase activity.
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Atorvastatin rapidly improves redox state in human vein grafts ex-vivo, by directly reducing NADPH-oxidase activity and improving eNOS coupling
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Myocardial Redox State Predicts Post-Operative Clinical Outcome in Patients Undergoing Elective CABG: The Role of NADPH-Oxidase and Uncoupled Nitric Oxide Synthase
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Short-Term Atorvastatin Treatment Reduces Vascular Oxidative Stress Saphenous Vein Grafts by Improving eNOS Coupling and Reducing NADPH-Oxidase Activity in Patients Undergoing Elective CABG
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