Cita APA (7th ed.)

Lloyd, S., Pearce, S., Günther, W., Kawaguchi, H., Igarashi, T., Jentsch, T., & Thakker, R. (1997). Idiopathic low molecular weight proteinuria associated with hypercalciuric nephrocalcinosis in Japanese children is due to mutations of the renal chloride channel (CLCN5).

Cita Chicago (17th ed.)

Lloyd, SE, S. Pearce, W. Günther, H. Kawaguchi, T. Igarashi, T. Jentsch, i R. Thakker. Idiopathic Low Molecular Weight Proteinuria Associated with Hypercalciuric Nephrocalcinosis in Japanese Children Is Due to Mutations of the Renal Chloride Channel (CLCN5). 1997.

Cita MLA (9th ed.)

Lloyd, SE, et al. Idiopathic Low Molecular Weight Proteinuria Associated with Hypercalciuric Nephrocalcinosis in Japanese Children Is Due to Mutations of the Renal Chloride Channel (CLCN5). 1997.

Atenció: Aquestes cites poden no estar 100% correctes.