Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.

ग्रंथसूची विवरण
मुख्य लेखकों:	Zhang, Y, Zhang, M, Sears, C, Emanuel, K, Redwood, C, El-Armouche, A, Kranias, E, Casadei, B
स्वरूप:	Journal article
भाषा:	English
प्रकाशित:	2008

Regulation of myocardial relaxation by neuronal NOS: Role phospholamban phosphorylation and phosphatase activity
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Cardiac nNOS regulates myocardial relaxation by stimulating phospholamban phosphorylation
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Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
द्वारा: Zhang, Y, और अन्य
प्रकाशित: (2005)

Reduced phospholamban phosphorylation is responsible for impaired myocardial relaxation in nNOS(-/-) mice
द्वारा: Zhang, Y, और अन्य
प्रकाशित: (2006)

Regulation of myocyte contractility by endogenous NO: role of protein phosphatase 2A and protein kinase A-dependent signalling
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Mechanical and electrophysiological properties of murine hypertrophied left ventricular myocytes
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Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
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Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
द्वारा: Sears, C, और अन्य
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
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प्रकाशित: (2003)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
द्वारा: Ashley, E, और अन्य
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The importance of the Thr17 residue of phospholamban as a phosphorylation site under physiological and pathological conditions
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Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
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nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
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Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
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Phospholamban R14del disease: The past, the present and the future
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AMP-Activated Protein Kinase Phosphorylates Cardiac Troponin I and Alters Contractility of Murine Ventricular Myocytes
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AMP-Activated protein kinase phosphorylates cardiac troponin i and alters contractility of murine ventricular myocytes
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AMP-activated protein kinase phosphorylates cardiac troponin I and alters contractility of murine ventricular myocytes.
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Blocking phospholamban with VHH intrabodies enhances contractility and relaxation in heart failure
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Obesity does not Lead to Imbalance Between Myocardial Phospholamban Phosphorylation and Dephosphorylation
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A novel role for phospholamban in the thalamic reticular nucleus
द्वारा: Benjamin Klocke, और अन्य
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
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Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
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A hallmark of phospholamban functional divergence is located in the N-terminal phosphorylation domain
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Myocardial nNOS-derived NO regulates excitation-contraction coupling via differential and cGMP-independent effects on protein phosphorylation
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Structural basis for allosteric control of the SERCA-Phospholamban membrane complex by Ca2+ and phosphorylation
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DISTINCTIVE ROLE FOR PROTEIN PHOSPHATASES AND PROTEIN KINASES IN THE REGULATION OF MYOCARDIAL CONTRACTION BY THE NEURONAL NITRIC OXIDE SYNTHASE
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Selective gene deletion of the neuronal nitric oxide synthase (nNOS) accelerates pathological left ventricular remodelling and functional deterioration after myocardial infarction (MI)
द्वारा: Dawson, D, और अन्य
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Acidosis in models of cardiac ventricular myocytes
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Beta 3-adrenoceptors modulate left ventricular relaxation in the rat heart via the NO-cGMP-PKG pathway.
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Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
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Discordant Calcium Transient and Action Potential Alternans in a Canine Left-Ventricular Myocyte
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Progressive Reduction in Myocyte Autophagy After Myocardial Infarction in Rabbits: Association with Oxidative Stress and Left Ventricular Remodeling
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Calcium Homeostasis in Ventricular Myocytes of Diabetic Cardiomyopathy
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प्रकाशित: (2020-01-01)

Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.

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