Th17 cells expressing KIR3DL2+ and responsive to HLA-B27 homodimers are increased in ankylosing spondylitis.
CD4 Th cells producing the proinflammatory cytokine IL-17 (Th17) have been implicated in a number of inflammatory arthritides including the spondyloarthritides. Th17 development is promoted by IL-23. Ankylosing spondylitis, the most common spondyloarthritis (SpA), is genetically associated with both...
المؤلفون الرئيسيون: | Bowness, P, Ridley, A, Shaw, J, Chan, A, Wong-Baeza, I, Fleming, M, Cummings, F, McMichael, A, Kollnberger, S |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
2011
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مواد مشابهة
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KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2010) -
KIR3DL2 INTERACTION WITH HLA-B27 PROMOTES THE SURVIVAL OF TH17 CELLS IN ANKYLOSING SPONDYLITIS
حسب: Kollnberger, S, وآخرون
منشور في: (2010) -
Th17 cells expressing KIR3DL2+are increased in ankylosing spondylitis
حسب: Ridley, A, وآخرون
منشور في: (2010) -
Th17 cells expressing KIR3DL2 and enriched for gut homing markers are increased in ankylosing spondylitis
حسب: Ridley, A, وآخرون
منشور في: (2011) -
Interaction of HLA-B27 homodimers with KIR3DL1 and KIR3DL2, unlike HLA-B27 heterotrimers, is independent of the sequence of bound peptide.
حسب: Kollnberger, S, وآخرون
منشور في: (2007)