Efficient adenoviral infection with IkappaB alpha reveals that macrophage tumor necrosis factor alpha production in rheumatoid arthritis is NF-kappaB dependent.

Ítems similars

• VEGF expression in human macrophages is NF-kappaB-dependent: studies using adenoviruses expressing the endogenous NF-kappaB inhibitor IkappaBalpha and a kinase-defective form of the IkappaB kinase 2.
  per: Kiriakidis, S, et al.
  Publicat: (2003)
• VEGF expression in human macrophages is NF-kappaB-dependent: studies using adenoviruses expressing the endogenous NF-kappaB inhibitor IkappaBalpha and a kinase-defective form of the IkappaB kinase 2.
  per: Kiriakidis, S, et al.
  Publicat: (2003)
• The TCL1A oncoprotein interacts directly with the NF-kappaB inhibitor IkappaB.
  per: Virginie Ropars, et al.
  Publicat: (2009-08-01)
• Effective adenoviral transfer of IkappaBalpha into human fibroblasts and chondrosarcoma cells reveals that the induction of matrix metalloproteinases and proinflammatory cytokines is nuclear factor-kappaB dependent.
  per: Bondeson, J, et al.
  Publicat: (2000)
• Selective regulation of cytokine induction by adenoviral gene transfer of IkappaBalpha into human macrophages: lipopolysaccharide-induced, but not zymosan-induced, proinflammatory cytokines are inhibited, but IL-10 is nuclear factor-kappaB independent.
  per: Bondeson, J, et al.
  Publicat: (1999)