NKT cells join the two step for inflammasome-independent IL-1β release
The cytokine interleukin-1β (IL-1β) is critical for antimicrobial defenses; the inflammasome pathway typically controls IL-1β release, but pathogens often evade this pathway. In this issue, Donado et al. (2020) describe an alternative, two-cell model, to instruct inflammasome-independent IL-1β relea...
Main Authors: | , |
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Format: | Journal article |
Language: | English |
Published: |
Cell Press
2020
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