Exploring neuroinflammatory processes in a mouse model of amyotrophic lateral sclerosis
<p>Amyotrophic lateral sclerosis (ALS) is a devastating degenerative disease, affecting both upper and lower motor neurons in the CNS. Around 10% of ALS cases are classed as familial, and around 20% of these are due to a mutation in the gene superoxide dismutase-1 (SOD1). The SOD1G93A mouse is...
Κύριοι συγγραφείς: | Evans, M, Mr Matthew C Evans |
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Άλλοι συγγραφείς: | Turner, M |
Μορφή: | Thesis |
Γλώσσα: | English |
Έκδοση: |
2012
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Θέματα: |
Παρόμοια τεκμήρια
Παρόμοια τεκμήρια
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The genetics of amyotrophic lateral sclerosis
ανά: Schymick, J
Έκδοση: (2009) -
In vitro transgenic models to elucidate the molecular mechanisms of TDP-43 pathology in amyotrophic lateral sclerosis
ανά: Mutihac, R
Έκδοση: (2013) -
Model systems for exploring new therapeutic interventions and disease mechanisms in spinal muscular atrophies (SMAs)
ανά: Sleigh, JN
Έκδοση: (2012) -
Investigating TDP43 biological dysfunction through the characterisation of Tardbp ENU mouse mutants: implications for neurodegeneration
ανά: De Sao Jose Martinho De Oliveira, H, κ.ά.
Έκδοση: (2014) -
Molecular mechanisms of OXR1 function
ανά: Liu, K
Έκδοση: (2014)