Neuronal nitric oxide synthase gene transfer promotes cardiac vagal gain of function.
Nitric oxide (NO) generated from neuronal nitric oxide synthase (NOS-1) in intrinsic cardiac ganglia has been implicated in parasympathetic-induced bradycardia. We provide direct evidence that NOS-1 acts in a site-specific manner to promote cardiac vagal neurotransmission and bradycardia. NOS-1 gene...
Main Authors: | , , , , , , |
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Format: | Journal article |
Language: | English |
Published: |
2002
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