Neuronal nitric oxide synthase gene transfer promotes cardiac vagal gain of function.
Nitric oxide (NO) generated from neuronal nitric oxide synthase (NOS-1) in intrinsic cardiac ganglia has been implicated in parasympathetic-induced bradycardia. We provide direct evidence that NOS-1 acts in a site-specific manner to promote cardiac vagal neurotransmission and bradycardia. NOS-1 gene...
Những tác giả chính: | Mohan, R, Heaton, D, Danson, E, Krishnan, S, Cai, S, Channon, K, Paterson, D |
---|---|
Định dạng: | Journal article |
Ngôn ngữ: | English |
Được phát hành: |
2002
|
Những quyển sách tương tự
-
Enhanced NOS-1 expression with gene transfer promotes cardiac vagal gain of function
Bằng: Mohan, R, et al.
Được phát hành: (2002) -
Enhanced neuronal nitric oxide synthase expression is central to cardiac vagal phenotype in exercise-trained mice.
Bằng: Danson, E, et al.
Được phát hành: (2003) -
Targeting neuronal nitric oxide synthase with gene transfer to modulate cardiac autonomic function.
Bằng: Mohan, R, et al.
Được phát hành: (2004) -
Targeting cardiac sympatho-vagal imbalance using gene transfer of nitric oxide synthase.
Bằng: Danson, E, et al.
Được phát hành: (2009) -
Gene transfer of neuronal nitric oxide synthase into intracardiac Ganglia reverses vagal impairment in hypertensive rats.
Bằng: Heaton, D, et al.
Được phát hành: (2007)