Tenascin-C modulates matrix contraction via focal adhesion kinase- and Rho-mediated signaling pathways.
A provisional matrix consisting of fibrin and fibronectin (FN) is deposited at sites of tissue damage and repair. This matrix serves as a scaffold for fibroblast migration into the wound where these cells deposit new matrix to replace lost or damaged tissue and eventually contract the matrix to brin...
| Main Authors: | , |
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| 格式: | Journal article |
| 語言: | English |
| 出版: |
2002
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