Tenascin-C modulates matrix contraction via focal adhesion kinase- and Rho-mediated signaling pathways.
A provisional matrix consisting of fibrin and fibronectin (FN) is deposited at sites of tissue damage and repair. This matrix serves as a scaffold for fibroblast migration into the wound where these cells deposit new matrix to replace lost or damaged tissue and eventually contract the matrix to brin...
Váldodahkkit: | Midwood, K, Schwarzbauer, J |
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Materiálatiipa: | Journal article |
Giella: | English |
Almmustuhtton: |
2002
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Geahča maid
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Tenascin-C suppresses Rho activation.
Dahkki: Wenk, M, et al.
Almmustuhtton: (2000) -
Coregulation of fibronectin signaling and matrix contraction by tenascin-C and syndecan-4.
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Almmustuhtton: (2004) -
Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease
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Almmustuhtton: (2018) -
Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease
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Almmustuhtton: (2017) -
Activation of Rho-kinase and focal adhesion kinase regulates the organization of stress fibers and focal adhesions in the central part of fibroblasts
Dahkki: Kazuo Katoh
Almmustuhtton: (2017-11-01)