Tenascin-C modulates matrix contraction via focal adhesion kinase- and Rho-mediated signaling pathways.
A provisional matrix consisting of fibrin and fibronectin (FN) is deposited at sites of tissue damage and repair. This matrix serves as a scaffold for fibroblast migration into the wound where these cells deposit new matrix to replace lost or damaged tissue and eventually contract the matrix to brin...
Автори: | Midwood, K, Schwarzbauer, J |
---|---|
Формат: | Journal article |
Мова: | English |
Опубліковано: |
2002
|
Схожі ресурси
Схожі ресурси
-
Tenascin-C suppresses Rho activation.
за авторством: Wenk, M, та інші
Опубліковано: (2000) -
Coregulation of fibronectin signaling and matrix contraction by tenascin-C and syndecan-4.
за авторством: Midwood, K, та інші
Опубліковано: (2004) -
Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease
за авторством: Giblin, S, та інші
Опубліковано: (2018) -
Alternative splicing of tenascin-C modulates cell-matrix interactions during inflammation and disease
за авторством: Giblin, S, та інші
Опубліковано: (2017) -
Activation of Rho-kinase and focal adhesion kinase regulates the organization of stress fibers and focal adhesions in the central part of fibroblasts
за авторством: Kazuo Katoh
Опубліковано: (2017-11-01)