MLP accumulation and remodelling in the infarcted rat heart.
Mutation of cytoskeletal protein genes results in abnormal protein function and causes cardiomyopathy. We hypothesised that cardiac levels of cytoskeletal proteins, such as dystrophin, desmin and muscle LIM protein (MLP), would be altered during remodelling caused by myocardial infarction (MI). We m...
Main Authors: | Wilding, JR, Lygate, C, Davies, K, Neubauer, S, Clarke, K |
---|---|
Format: | Journal article |
Language: | English |
Published: |
2006
|
Similar Items
-
Dystrophin- and MLP-deficient mouse hearts: marked differences in morphology and function, but similar accumulation of cytoskeletal proteins.
by: Wilding, JR, et al.
Published: (2005) -
Mitochondrial abnormalities in chronically infarcted rat heart
by: Murray, A, et al.
Published: (2004) -
The PPARgamma-activator rosiglitazone does not alter remodeling but increases mortality in rats post-myocardial infarction.
by: Lygate, C, et al.
Published: (2003) -
Insulin resistance, abnormal energy metabolism and increased ischemic damage in the chronically infarcted rat heart.
by: Murray, A, et al.
Published: (2006) -
CINE-MR imaging of the normal and infarcted rat heart using an 11.7 T vertical bore MR system.
by: Tyler, D, et al.
Published: (2006)