BEHAVIORAL RECOVERY IN RATS WITH UNILATERAL 6-OHDA LESIONS FOLLOWING IMPLANTATION OF NIGRAL CELL-SUSPENSIONS IN DIFFERENT FOREBRAIN SITES

BEHAVIORAL RECOVERY IN RATS WITH UNILATERAL 6-OHDA LESIONS FOLLOWING IMPLANTATION OF NIGRAL CELL-SUSPENSIONS IN DIFFERENT FOREBRAIN SITES

Xehetasun bibliografikoak
Egile Nagusiak:	Dunnett, S, Bjorklund, A, Schmidt, R, Stenevi, U, Iversen, S
Formatua:	Journal article
Argitaratua:	1983

Antzeko izenburuak

BEHAVIORAL RECOVERY IN RATS WITH BILATERAL 6-OHDA LESIONS FOLLOWING IMPLANTATION OF NIGRAL CELL-SUSPENSIONS
nork: Dunnett, S, et al.
Argitaratua: (1983)

Behavioural recovery following transplantation of substantia nigra in rats subjected to 6-OHDA lesions of the nigrostriatal pathway. I. Unilateral lesions.
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Behavioural recovery following transplantation of substantia nigra in rats subjected to 6-OHDA lesions of the nigrostriatal pathway. II. Bilateral lesions.
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Functional reactivation of the deafferented neostriatum by nigral transplants.
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Grafts of embryonic substantia nigra reinnervating the ventrolateral striatum ameliorate sensorimotor impairments and akinesia in rats with 6-OHDA lesions of the nigrostriatal pathway.
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Nigral transplants reinnervating the dopamine-depleted neostriatum can sustain intracranial self-stimulation.
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Regulatory impairments following selective 6-OHDA lesions of the neostriatum.
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CNS transplantation: structural and functional recovery from brain damage.
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Survival of Nigral Grafts within the Striatum of Marmosets with 6-Ohda Lesions Depends Critically on Donor Embryo Age
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Septal transplants restore maze learning in rats with fornix-fimbria lesions.
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Function recovery following neural transplantation of embryonic septal nuclei in adult rats with septohippocampal lesions.
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HIPPOCAMPAL DEAFFERENTATION - TRANSPLANT DERIVED REINNERVATION AND FUNCTIONAL RECOVERY
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HIPPOCAMPAL DEAFFERENTATION - TRANSPLANT-DERIVED REINNERVATION AND FUNCTIONAL RECOVERY
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Transplantation of embryonic ventral forebrain neurons to the neocortex of rats with lesions of nucleus basalis magnocellularis--I. Biochemical and anatomical observations.
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Transplantation of embryonic ventral forebrain neurons to the neocortex of rats with lesions of nucleus basalis magnocellularis--II. Sensorimotor and learning impairments.
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Cholinergic ventral forebrain grafts into the neocortex improve passive avoidance memory in a rat model of Alzheimer disease.
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Reinnervation of the denervated striatum by substantia nigra transplants: functional consequences as revealed by pharmacological and sensorimotor testing.
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Transplantation of embryonic ventral forebrain grafts to the neocortex of rats with bilateral lesions of nucleus basalis magnocellularis ameliorates a lesion-induced deficit in spatial memory.
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Human decisions about when to act originate within a basal forebrain–nigral circuit
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Ascorbic Acid Increases the Number of Dopamine Neurons In Vitro and in Transplants to the 6-OHDA-Lesioned Rat Brain
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The modulatory effect of 100 Hz electroacupuncture on striatal synaptic plasticity in unilateral lesioned 6-OHDA rats
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Effects of Surgical Anaesthesia on the Viability of Nigral Grafts in the Rat Striatum
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Time dependent degeneration of the nigrostriatal tract in mice with 6-OHDA lesioned medial forebrain bundle and the effect of activin A on l-Dopa induced dyskinesia
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Polysomnographic Features of Sleep Disturbances and REM Sleep Behavior Disorder in the Unilateral 6-OHDA Lesioned Hemiparkinsonian Rat
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Amphetamine and apomorphine responses in the rat following 6-OHDA lesions of the nucleus accumbens septi and corpus striatum.
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Dissociable effects of 6-OHDA-induced lesions of neostriatum on anorexia, locomotor activity and stereotypy: the role of behavioural competition.
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RGS4 is involved in the generation of abnormal involuntary movements in the unilateral 6-OHDA-lesioned rat model of Parkinson's disease
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Contributive Role of TNF-α to L-DOPA-Induced Dyskinesia in a Unilateral 6-OHDA Lesion Model of Parkinson’s Disease
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Learning in the absence of forebrain noradrenaline.
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Mineralization of the fascicula nigrale
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Regulatory impairments following selective kainic acid lesions of the neostriatum.
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Pioglitazone enhances brain mitochondrial biogenesis and phase II detoxification capacity in neonatal rats with 6-OHDA-induced unilateral striatal lesions
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