ENDOTHELIAL SPECIFIC NOX2 OVER-EXPRESSION INCREASES SUSCEPTIBILITY TO ANGIOTENSIN II INDUCED AORTIC DISSECTION

Similar Items

• Targeted endothelial Nox2 over-expression potentiates angiotensin ii induced aortic dissection in mice
  by: Fan, L, et al.
  Published: (2010)
• Endothelial Specific Redox Signalling Regulates Susceptibility to Aortic Dissection Through Endothelial Cyclophilin A secretion
  by: Fan, L, et al.
  Published: (2011)
• Endothelial cell-specific reactive oxygen species production increases susceptibility to aortic dissection
  by: Fan, L, et al.
  Published: (2014)
• Endothelial cell-specific reactive oxygen species production increases susceptibility to aortic dissection.
  by: Fan, L, et al.
  Published: (2014)
• Deficiency in endothelial cell tetrahydrobiopterin increases resistance vascular remodelling, blood pressure, and susceptibility to aortic abdominal aneurysm in response to angiotensin II
  by: Chuaiphichai, S, et al.
  Published: (2018)