THE ROLE OF HLA-B27 KIR3DL2 INTERACTIONS IN THE PATHOGENESIS OF SPONDYLOARTHRITIS

THE ROLE OF HLA-B27 KIR3DL2 INTERACTIONS IN THE PATHOGENESIS OF SPONDYLOARTHRITIS

Podrobná bibliografie
Hlavní autoři:	Kollnberger, S, Shaw, J, Ridley, A, Wong-Baeza, I, McHugh, K, Keidel, S, Chan, A, Bowness, P
Médium:	Conference item
Vydáno:	2012

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The arthritis-associated HLA-B*27:05 allele forms more cell surface B27 dimer and free heavy chain ligands for KIR3DL2 than HLA-B*27:09.
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Th17 cells expressing KIR3DL2+are increased in ankylosing spondylitis
Autor: Ridley, A, a další
Vydáno: (2010)

Factors determining the cell surface expression of a novel HLA-B27 epitope
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Vydáno: (2010)

HLA-B27 FORMS HEAVY CHAIN HOMODIMERS IN VITRO AND IN VIVO THAT BIND TO CELLULAR RECEPTORS INCLUDING LILR AND KIR FAMILY MEMBERS
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Vydáno: (2010)

TH17 CELLS EXPRESSING KIR3DL2 AND ENRICHED FOR GUT HOMING MARKERS ARE INCREASED IN ANKYLOSING SPONDYLITIS
Autor: Ridley, A, a další
Vydáno: (2012)

HLA-B27 heavy chain homodimers are expressed in HLA-B27 transgenic rodent models of spondyloarthritis and are ligands for paired Ig-like receptors.
Autor: Kollnberger, S, a další
Vydáno: (2004)

Direct visualisation of HLA-B27 forms in cell lines and Spondyloarthropathy tissues
Autor: Rysnik, O, a další
Vydáno: (2013)

HLA-B*2709, A SUBTYPE NOT ASSOCIATED WITH ANKYLOSING SPONDYLITIS, FORMS FEWER HOMODIMERS THAN HLA-B*2705
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Vydáno: (2010)

Supplementary figures for "Inhibiting HLA-B27 homdimer-driven immune cell inflammation in Spondyloarthritis"
Autor: Bowness, P
Vydáno: (2012)

Increased IL-23 receptor expression is observed on KIR3DL2+CD4+T cells in Ankylosing Spondylitis and correlates with IL-23R polymorphisms
Autor: Ridley, A, a další
Vydáno: (2013)