Using an engineered GluCl channel to silence sensory neurons and treat neuropathic pain at the source
Peripheral neuropathic pain arises as a consequence of injury to sensory neurons; the development of ectopic activity in these neurons is thought to be critical for the induction and maintenance of such pain. Local anaesthetics and anti-epileptic drugs can suppress hyper-excitability however these d...
| Main Authors: | , , , , , , |
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| 格式: | Journal article |
| 出版: |
Oxford University Press
2017
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