CD66a interactions between human melanoma and NK cells: a novel class I MHC-independent inhibitory mechanism of cytotoxicity.
NK cells are able to kill virus-infected and tumor cells via a panel of lysis receptors. Cells expressing class I MHC proteins are protected from lysis primarily due to the interactions of several families of NK receptors with both classical and nonclassical class I MHC proteins. In this study we sh...
Main Authors: | Markel, G, Lieberman, N, Katz, G, Arnon, T, Lotem, M, Drize, O, Blumberg, R, Bar-Haim, E, Mader, R, Eisenbach, L, Mandelboim, O |
---|---|
格式: | Journal article |
语言: | English |
出版: |
2002
|
相似书籍
-
MHC class I-independent recognition of NK-activating receptor KIR2DS4.
由: Katz, G, et al.
出版: (2004) -
Recognition of HLA-Cw4 but not HLA-Cw6 by the NK cell receptor killer cell Ig-like receptor two-domain short tail number 4.
由: Katz, G, et al.
出版: (2001) -
Enhanced recognition of human NK receptors after influenza virus infection.
由: Achdout, H, et al.
出版: (2003) -
Carcinoembryonic antigen (CEA) inhibits NK killing via interaction with CEA-related cell adhesion molecule 1.
由: Stern, N, et al.
出版: (2005) -
The mechanisms controlling NK cell autoreactivity in TAP2-deficient patients.
由: Markel, G, et al.
出版: (2004)