Zoledronic acid inhibits the development of osteolytic bone disease and increases disease free survival in a murine model of multiple myeloma.

Zoledronic acid inhibits the development of osteolytic bone disease and increases disease free survival in a murine model of multiple myeloma.

Xehetasun bibliografikoak
Egile Nagusiak:	Croucher, P, Hijzen, A, Shipman, C, Perry, M, Lippitt, J, Green, J, Van Camp, B, Vanderkerken, K
Formatua:	Journal article
Argitaratua:	2001

Antzeko izenburuak

Zoledronic acid prevents the development of osteolytic bone disease and increases survival in a murine model of multiple myeloma
nork: Croucher, P, et al.
Argitaratua: (2002)

Zoledronic acid prevents the development of myeloma bone disease and increases survival
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Osteoprotegerin inhibits the development of osteolytic bone disease in multiple myeloma.
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Osteoprotegerin inhibits the development of osteolytic bone disease in multiple myeloma
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Zoledronic acid treatment of 5T2MM-bearing mice inhibits the development of myeloma bone disease: evidence for decreased osteolysis, tumor burden and angiogenesis, and increased survival.
nork: Croucher, P, et al.
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Osteoprotegerin (OPG) inhibits the development of osteolytic bone disease in the 5T2MM model of multiple myeloma.
nork: Croucher, P, et al.
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Bisphosphonates and osteoprotegerin as inhibitors of myeloma bone disease.
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Osteoprotegerin and the development of myeloma bone disease
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The potent bisphosphonate ibandronate does not induce myeloma cell apoptosis in a murine model of established multiple myeloma.
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Myeloma cells can down-regulate release of osteoprotegerin (OPG) from human bone cells - A contributing factor in myeloma bone loss?
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Recombinant osteoprotegerin decreases tumor burden and increases survival in a murine model of multiple myeloma.
nork: Vanderkerken, K, et al.
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Bone marrow endothelial cells produce osteoprotegerin: Evidence for a role in the development of myeloma bone disease
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Bone marrow endothelial cells express the osteoclastogenic factor RANKL and its decoy receptor OPG: Evidence for a role in the development of myeloma bone disease.
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APOMINE (TM), a novel inhibitor of the meyalonate/isoprenoid pathway, promotes apoptosis of myeloma cells in vitro and is associated with a modulation of the myeloma disease in vivo.
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Bisphosphonates and in vivo models of multiple myeloma - Reply
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Myeloma cells isolated from patients with multiple myeloma express osteoprotegerin ligand.
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Osteocyte CIITA aggravates osteolytic bone lesions in myeloma
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CST6 suppresses osteolytic bone disease in multiple myeloma by blocking osteoclast differentiation
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Bortezomib-releasing silica-collagen xerogels for local treatment of osteolytic bone- and minimal residual disease in multiple myeloma
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Bisphosphonates--mechanisms of action in multiple myeloma.
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A loss of host-derived MMP-7 promotes myeloma growth and osteolytic bone disease in vivo
nork: Lwin, S, et al.
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Correction: Bortezomib-releasing silica-collagen xerogels for local treatment of osteolytic bone- and minimal residual disease in multiple myeloma
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Tumour cells isolated from patients with multiple myeloma express the critical osteoclastogenic factor, RANKL.
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Effect of glycosphingolipids on osteoclastogenesis and osteolytic bone diseases.
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Myeloma cells can promote osteoclastic activity rather than osteoclast recruitment in vitro; Evidence from the 5T2MM murine model of myeloma
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Elucidation of molecular basis of osteolytic bone lesions in advanced multiple myeloma
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5T2MM murine myeloma cells and CD138+ve human myeloma cells promote osteoclastic activity rather than osteoclast formation in vitro
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Advances in the management of myeloma bone disease.
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Osteolytic disease in IL-6 and Myc dependent mouse model of human myeloma
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LONG-ACTING ZOLEDRONIC ACID: ONCE-YEARLY ADMINISTRATION AND EFFICACY EVALUATION IN MYELOMA BONE DISEASE
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Osteolytic lesions: Multiple myeloma or prostate cancer?
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Preventing osteolytic lesions and osteomyelitis in multiple myeloma
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Tumor-host cell interactions in the bone disease of myeloma.
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Promotion of Bone Lesions Through the Myeloma Integrin α6-Mediated Osteolytic Signaling
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Soluble rank ligand produced by myeloma cells causes generalised bone loss in multiple myeloma.
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The bisphosphonate, zoledronic acid, induces apoptosis of breast cancer cells: evidence for synergy with paclitaxel.
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EZH2 serves as a viable therapeutic target for myeloma-induced osteolytic bone destruction
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Multiple myeloma presenting as a massive osteolytic lesion at the clivus
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Osteoprotegerin is a soluble decoy receptor for tumor necrosis factor-related apoptosis-inducing ligand/Apo2 ligand and can function as a paracrine survival factor for human myeloma cells.
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Role of Tumor-Derived Chemokines in Osteolytic Bone Metastasis
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