Normal radial migration and lamination are maintained in dyslexia-susceptibility candidate gene homolog Kiaa0319 knockout mice.
Developmental dyslexia is a common disorder with a strong genetic component, but the underlying molecular mechanisms are still unknown. Several candidate dyslexia susceptibility genes, including KIAA0319, DYX1C1 and DCDC2, have been identified in humans. RNA interference experiments targeting these...
المؤلفون الرئيسيون: | Martinez-Garay, I, Guidi, L, Holloway, Z, Bailey, M, Lyngholm, D, Schneider, T, Donnison, T, Butt, S, Velayos-Baeza, A, Molnár, Z, Monaco, A |
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التنسيق: | Journal article |
اللغة: | English |
منشور في: |
Springer
2016
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مواد مشابهة
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Knockout mice for dyslexia susceptibility gene homologs KIAA0319 and
KIAA0319L have unaffected neuronal migration but display abnormal
auditory processing
حسب: Guidi, L, وآخرون
منشور في: (2017) -
Alternative splicing in the dyslexia-associated gene KIAA0319.
حسب: Velayos-Baeza, A, وآخرون
منشور في: (2007) -
Functional characterisation of dyslexia-susceptibility candidate genes KIAA0319 and KIAA0319L
حسب: Guidi, L
منشور في: (2017) -
The dyslexia-associated KIAA0319 protein undergoes proteolytic processing with {gamma}-secretase-independent intramembrane cleavage.
حسب: Velayos-Baeza, A, وآخرون
منشور في: (2010) -
The dyslexia-associated protein KIAA0319 interacts with adaptor protein 2 and follows the classical clathrin-mediated endocytosis pathway.
حسب: Levecque, C, وآخرون
منشور في: (2009)