Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes

ग्रंथसूची विवरण
मुख्य लेखकों:	Zhang, Y, Sears, C, Ashley, E, Kim, Y, Casadei, B
स्वरूप:	Conference item
प्रकाशित:	2003

Cardiac nitric oxide synthase 1 regulates basal and beta-adrenergic contractility in murine ventricular myocytes.
द्वारा: Ashley, E, और अन्य
प्रकाशित: (2002)

A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
द्वारा: Sears, C, और अन्य
प्रकाशित: (2002)

Enhancement of beta adrenergic responsiveness in myocytes lacking nitric oxide synthase 1
द्वारा: Ashley, E, और अन्य
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Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left-ventricular contraction and relaxation in vivo
द्वारा: Ashley, E, और अन्य
प्रकाशित: (2003)

Nitric oxide synthase 1 (NOS1) regulates basal and beta-adrenergic left ventricular contraction and relaxation in vivo
द्वारा: Ashley, E, और अन्य
प्रकाशित: (2003)

nNOS V eNOS mediated regulation of beta adrenergic inotropic and lusitropic responses in left ventricular myocytes
द्वारा: Zhang, Y, और अन्य
प्रकाशित: (2004)

Cardiac neuronal nitric oxide synthase isoform regulates myocardial contraction and calcium handling.
द्वारा: Sears, C, और अन्य
प्रकाशित: (2003)

Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
द्वारा: Sears, C, और अन्य
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Mechanical and electrophysiological properties of murine hypertrophied left ventricular myocytes
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NEURONAL NITRIC OXIDE SYNTHASE-MEDIATED REGULATION OF MYOCARDIAL REDOX STATE AND beta 3 ADRENERGIC RECEPTOR RESPONSES
द्वारा: Idigo, W, और अन्य
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A cardiac nitric oxide synthase 1 regulates contraction and calcium fluxes - a novel mechanism in the autocrine control of cardiac function
द्वारा: Ashley, E, और अन्य
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Impaired beta-adrenergic response and decreased L-type calcium current of hypertrophied left ventricular myocytes in postinfarction heart failure
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
द्वारा: Bryant, S, और अन्य
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
द्वारा: Sears, C, और अन्य
प्रकाशित: (2000)

MODELLING CALCIUM HANDLING MECHANISMS IN NORMAL AND NEURONAL NITRIC OXIDE SYNTHASE KNOCK-OUT MOUSE HEARTS
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Myocardial NOS1 controls the lusitropic response to beta-adrenergic stimulation in vivo and in vitro
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Nitric oxide control of cardiac function: is neuronal nitric oxide synthase a key component?
द्वारा: Sears, C, और अन्य
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Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.
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Superoxide production by left ventricular myocytes in control and hypertrophied hearts
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Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation
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Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
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Modelling of calcium handling in airway myocytes.
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Calcium handling by vascular myocytes in hypertension
द्वारा: R.C.A. Tostes, और अन्य
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Effects of NAADP on contraction and calcium transients in guinea-pig isolated ventricular myocytes
द्वारा: Macgregor, A, और अन्य
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Are myocardial eNOS and nNOS involved in the beta-adrenergic and muscarinic regulation of inotropy? A systematic investigation.
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Mechanical and electrophysiological properties of left ventricular myocytes in catecholamine-induced hypertrophy in mice
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Impairment of cholinergic signaling reduces atrial beta-adrenergic responsiveness by enhancing eNOS expression
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Effect of disruption of the nNOS gene on myocardial calcium current and contraction
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NEURONAL NITRIC OXIDE SYNTHASE GENE DISRUPTION REDUCES THE DIASTOLIC CALCIUM LEAK FROM THE RYANODINE RECEPTOR: IMPLICATIONS FOR EXCITATION-CONTRACTION COUPLING IN HEART FAILURE
द्वारा: Lim, G, और अन्य
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Actions of a membrane permeant analogue of NAADP on contractions and calcium sparks in cardiac ventricular myocytes.
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Calcium Homeostasis in Ventricular Myocytes of Diabetic Cardiomyopathy
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Cardiac myocyte intrinsic contractility and calcium handling deficits underlie heart organ dysfunction in murine cancer cachexia
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A mathematical model of the mouse ventricular myocyte contraction.
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Disruption of inhibitory G-proteins mediates a reduction in atrial beta-adrenergic signaling by enhancing eNOS expression.
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DISTINCTIVE ROLE FOR PROTEIN PHOSPHATASES AND PROTEIN KINASES IN THE REGULATION OF MYOCARDIAL CONTRACTION BY THE NEURONAL NITRIC OXIDE SYNTHASE
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AMP-Activated protein kinase phosphorylates cardiac troponin i and alters contractility of murine ventricular myocytes
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AMP-activated protein kinase phosphorylates cardiac troponin I and alters contractility of murine ventricular myocytes.
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A compartmentalized mathematical model of the β1-adrenergic signaling system in mouse ventricular myocytes.
द्वारा: Vladimir E Bondarenko
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Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes

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