SMAD2/3-SMYD2 and developmental transcription factors cooperate with cell cycle inhibitors to guide tissue formation
Tissue formation and organ homeostasis are achieved by precise coordination of proliferation and differentiation of stem cells and progenitors. While deregulation of these processes can result in degenerative disease or cancer, their molecular interplays remain unclear. Here we show that the switch...
Auteurs principaux: | Militi, S, Nibhani, R, Pook, M, Pauklin, S |
---|---|
Format: | Journal article |
Langue: | English |
Publié: |
Oxford University Press
2024
|
Documents similaires
-
RBL2-E2F-GCN5 guide cell fate decisions during tissue specification by regulating cell-cycle-dependent fluctuations of non-cell-autonomous signaling
par: Militi, S, et autres
Publié: (2023) -
RBL2-E2F-GCN5 guide cell fate decisions during tissue specification by regulating cell-cycle-dependent fluctuations of non-cell-autonomous signaling
par: Stefania Militi, et autres
Publié: (2023-09-01) -
Epigenetic regulation of SMAD3 by histone methyltransferase SMYD2 promotes lung cancer metastasis
par: Kwangho Kim, et autres
Publié: (2023-05-01) -
BRD9-SMAD2/3 orchestrates stemness and tumorigenesis in pancreatic ductal adenocarcinoma
par: Feng, Y, et autres
Publié: (2023) -
SMYD3 induces sorafenib resistance by activating SMAD2/3-mediated epithelial-mesenchymal transition in hepatocellular carcinoma
par: Shanshan Wang, et autres
Publié: (2023-07-01)