Using fluorescence emission spectroscopy to asses the Ca2+ affinity of reconstituted troponin and thin filaments containing cardiomyopathy causing mutations of troponin and alpha-tropomyosin

Samankaltaisia teoksia

• Dilated and hypertrophic cardiomyopathy mutations in troponin and alpha-tropomyosin have opposing effects on the calcium affinity of cardiac thin filaments.
  Tekijä: Robinson, P, et al.
  Julkaistu: (2007)
• Hypertrophic and dilated cardiomyopathycausing mutations of troponin and alpha-tropomyosin have opposite effects on thin filament calcium binding
  Tekijä: Robinson, P, et al.
  Julkaistu: (2007)
• Functional effects of mutations in troponin T and tropomyosin which cause dilated cardiomyopathy
  Tekijä: Robinson, P, et al.
  Julkaistu: (2003)
• Dilated cardiomyopathy causing mutations in cardiac troponin T (TNT), cardiac troponin C (TNC), and alpha tropomyosin (TM) reduce CA(2+) sensitivity and maximum calcium activation
  Tekijä: Redwood, C, et al.
  Julkaistu: (2004)
• Mutations in fast skeletal troponin I, troponin T and beta-tropomyosin that cause distal arthrogryposis all enhance thin filament activation
  Tekijä: Robinson, P, et al.
  Julkaistu: (2005)