Superoxide production by left ventricular myocytes in control and hypertrophied hearts

Superoxide production by left ventricular myocytes in control and hypertrophied hearts

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Bryant, S, Sears, C, Kim, Y, Channon, K, Casadei, B
التنسيق:	Conference item
منشور في:	2002

مواد مشابهة

Mechanical and electrophysiological properties of murine hypertrophied left ventricular myocytes
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Cardiomyocyte-Targeted Overexpression of GTP Cyclohydrolase-1 Increases Myocyte Lusitropy and Reduces Superoxide Production
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
حسب: Bryant, S, وآخرون
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Does nitric oxide regulate the hyperpolarisation-activated inward current in ventricular myocytes from spontaneously hypertensive rats?
حسب: Sears, C, وآخرون
منشور في: (2000)

Impaired beta-adrenergic response and decreased L-type calcium current of hypertrophied left ventricular myocytes in postinfarction heart failure
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Reduced phospholamban phosphorylation is associated with impaired relaxation in left ventricular myocytes from neuronal NO synthase-deficient mice.
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Nitric oxide does not modulate the hyperpolarization-activated current, I(f), in ventricular myocytes from spontaneously hypertensive rats.
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منشور في: (2001)

Electrophysiological and mechanical properties of ventricular myocytes isolated from wild-type mice and mice with disruption of the neuronal nitric oxide synthase gene
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Neuronal nitric oxide synthase (NOS1) regulates beta-adrenergic contraction and calcium handling in isolated murine ventricular myocytes
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
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A cardiac neuronal nitric oxide synthase isoform regulates contraction and calcium handling in murine ventricular myocytes
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