Molecular mechanisms underlying the myocardial nitric oxide-redox imbalance in human atrial fibrillation

Molecular mechanisms underlying the myocardial nitric oxide-redox imbalance in human atrial fibrillation

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριοι συγγραφείς:	Reilly, SN, Channon, K, Alp, N, Casadei, B
Μορφή:	Journal article
Έκδοση:	2009

Παρόμοια τεκμήρια

Molecular Basis of Atrial Nitroso-Redox Imbalance in Permanent Human Atrial Fibrillation
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Time-Dependent and Regional Differences in Myocardial Nitric Oxide-Redox State in Atrial Fibrillation
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Mechanisms regulating myocardial redox state in chronic atrial fibrillation: the role of NADPH-oxidase and uncoupled nitric oxide synthases
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Different sources of oxidant stress in the early and late stage of atrial fibrillation
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Myocardial Redox State Predicts Post-Operative Atrial Fibrillation in Cardiac Surgery
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Time-dependent regional differences in sources of oxidative stress in atrial fibrillation-induced remodelling
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Is increased myocardial oxidative stress an early event in the natural history of atrial fibrillation?
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NEURONAL NITRIC OXIDE SYNTHASE-MEDIATED REGULATION OF MYOCARDIAL REDOX STATE AND beta 3 ADRENERGIC RECEPTOR RESPONSES
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Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins.
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Nitric Oxide synthases and atrial fibrillation
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ATRIAL SOURCES OF REACTIVE OXYGEN SPECIES VARY WITH THE SUBSTRATE AND DURATION OF ATRIAL FIBRILLATION: IMPLICATIONS FOR THE ANTIARRHYTHMIC EFFECT OF STATINS
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P644Mechanisms of myocardial nitroso redox imbalance following elective cardiac surgery on cardiopulmonary bypass.
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ανά: Simon, J, κ.ά.
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Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins
ανά: Reilly, S, κ.ά.
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Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins.
ανά: Reilly, S, κ.ά.
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Tetrahydrobiopterin: coupling nitric oxide and redox signalling in cardiovascular disease
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Myocardial superoxide and peroxynitrite as strong independent predictors of paroxysmal atrial fibrillation post-CABG
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ανά: Carnicer, R, κ.ά.
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
ανά: Kim, Y, κ.ά.
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
ανά: Kim, Y, κ.ά.
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation.
ανά: Kim, Y, κ.ά.
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MYOCARDIAL O2-AND ONOO-GENERATION IN CHRONIC ATRIAL FIBRILLATION
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation (AF)
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The emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.
ανά: Casadei, B
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Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
ανά: Alp, N, κ.ά.
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Molecular mechanisms underlying the development of Atrial Fibrillation
ανά: Stefanini, M
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Electrolyte’s imbalance role in atrial fibrillation: Pharmacological management
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Rapid regulation of platelet activation in vivo by nitric oxide
ανά: Schafer, A, κ.ά.
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Multi-scale modelling and simulations into the mechanisms linking neuronal nitric oxide synthase and atrial fibrillation
ανά: Muszkiewicz, A
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase
ανά: Reilly, S, κ.ά.
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.
ανά: Reilly, S, κ.ά.
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EPR quantification of vascular nitric oxide production in genetically modified mouse models.
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Myocardial fibrosis and atrial fibrillation
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154 Myocardial nox2 activity regulates atrial fibrillation susceptibility
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