Molecular mechanisms underlying the myocardial nitric oxide-redox imbalance in human atrial fibrillation

Molecular mechanisms underlying the myocardial nitric oxide-redox imbalance in human atrial fibrillation

Manylion Llyfryddiaeth
Prif Awduron:	Reilly, SN, Channon, K, Alp, N, Casadei, B
Fformat:	Journal article
Cyhoeddwyd:	2009

Eitemau Tebyg

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Time-Dependent and Regional Differences in Myocardial Nitric Oxide-Redox State in Atrial Fibrillation
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Mechanisms regulating myocardial redox state in chronic atrial fibrillation: the role of NADPH-oxidase and uncoupled nitric oxide synthases
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Is increased myocardial oxidative stress an early event in the natural history of atrial fibrillation?
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Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins.
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ATRIAL SOURCES OF REACTIVE OXYGEN SPECIES VARY WITH THE SUBSTRATE AND DURATION OF ATRIAL FIBRILLATION: IMPLICATIONS FOR THE ANTIARRHYTHMIC EFFECT OF STATINS
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P644Mechanisms of myocardial nitroso redox imbalance following elective cardiac surgery on cardiopulmonary bypass.
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Compromised redox homeostasis, altered nitroso-redox balance and therapeutic possibilities in atrial fibrillation
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Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins
gan: Reilly, S, et al.
Cyhoeddwyd: (2011)

Atrial sources of reactive oxygen species vary with the duration and substrate of atrial fibrillation: implications for the antiarrhythmic effect of statins.
gan: Reilly, S, et al.
Cyhoeddwyd: (2011)

Tetrahydrobiopterin: coupling nitric oxide and redox signalling in cardiovascular disease
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Myocardial superoxide and peroxynitrite as strong independent predictors of paroxysmal atrial fibrillation post-CABG
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation
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A myocardial Nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation.
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MYOCARDIAL O2-AND ONOO-GENERATION IN CHRONIC ATRIAL FIBRILLATION
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A myocardial nox2 containing NAD(P)H oxidase contributes to oxidative stress in human atrial fibrillation (AF)
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Myocardial Redox State Predicts Post-Operative Clinical Outcome in Patients Undergoing Elective CABG: The Role of NADPH-Oxidase and Uncoupled Nitric Oxide Synthase
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The emerging role of neuronal nitric oxide synthase in the regulation of myocardial function.
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Regulation of endothelial nitric oxide synthase by tetrahydrobiopterin in vascular disease.
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Molecular mechanisms underlying the development of Atrial Fibrillation
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Rapid regulation of platelet activation in vivo by nitric oxide
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Multi-scale modelling and simulations into the mechanisms linking neuronal nitric oxide synthase and atrial fibrillation
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase
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Up-regulation of miR-31 in human atrial fibrillation begets the arrhythmia by depleting dystrophin and neuronal nitric oxide synthase.
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EPR quantification of vascular nitric oxide production in genetically modified mouse models.
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Myocardial fibrosis and atrial fibrillation
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154 Myocardial nox2 activity regulates atrial fibrillation susceptibility
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Regulation of myocyte function by gch1 overexpression mediated increase in nitric oxide
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Molecular chaperones and proteostasis regulation during redox imbalance
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Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin-mediated endothelial nitric oxide synthase coupling.
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Rapid, direct effects of statin treatment on arterial redox state and nitric oxide bioavailability in human atherosclerosis via tetrahydrobiopterin- mediated endothelial nitric oxide synthase coupling
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