Oxidative stress via activating soluble guanylate cyclase leads to increased nitric oxide sensitivity of coronary arterioles in high fat diet-induced obesity

Oxidative stress via activating soluble guanylate cyclase leads to increased nitric oxide sensitivity of coronary arterioles in high fat diet-induced obesity

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Erdei, N, Feher, A, Beleznai, T, Edes, I, Koller, A, Bagi, Z
التنسيق:	Conference item
منشور في:	2007

مواد مشابهة

Increased soluble guanylate cyclase (sGC) activity may compensate for the high fat diet-induced reduction in NO bioavailability of rat coronary arterioles
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Alteration of the soluble guanylate cyclase system in coronary arteries of high cholesterol diet‐fed rabbits
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Flow-induced constriction in arterioles of hyperhomocysteinemic rats is due to impaired nitric oxide and enhanced thromboxane A(2) mediation.
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Crystal structures of the catalytic domain of human soluble guanylate cyclase.
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Hydrogen peroxide elicits COX-2-dependent production of constrictor prostaglandins in arterioles of type 2 diabetic mice
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Obesity-induced reduction in nitric oxide-dependent arteriolar dilation in rats. Role of xanthine oxidase-derived superoxide.
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The levosimendan metabolite OR-1896 elicits vasodilation by activating the K(ATP) and BK(Ca) channels in rat isolated arterioles.
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The levosimendan metabolite OR-1896 elicits vasodilation by activating the K(ATP) and BK(Ca) channels in rat isolated arterioles.
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