BIM is a novel regulator of glucocorticoid-induced apoptosis in osteoblasts

BIM is a novel regulator of glucocorticoid-induced apoptosis in osteoblasts

Sonraí bibleagrafaíochta
Príomhchruthaitheoirí:	Espina, B, Russell, R, Hulley, P
Formáid:	Conference item
Foilsithe / Cruthaithe:	2006

Míreanna comhchosúla

Regulation of bim in glucocorticoid-mediated osteoblast apoptosis.
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Bim is transcriptionally regulated during induction of apoptosis by growth factor withdrawal in osteoblasts
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Bim as a potential target in osteoblast survival
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Bim, Bak, and Bax regulate osteoblast survival.
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The BH3-only protein BIM is upregulated in osteoblasts in response to dexamethasone treatment, growth factor deprivation and anoikis
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Vanadate prevents glucocorticoid-induced apoptosis of osteoblasts in vitro and osteocytes in vivo.
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The identification of transcriptional targets associated with osteoblast apoptosis signaling in response to growth factor withdrawal
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Sulforaphane reverses glucocorticoid-induced apoptosis in osteoblastic cells through regulation of the Nrf2 pathway
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Vanadate protects osteoblasts in vitro and in vivo from glucocorticoid impairment of proliferation
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The tyrosine phosphatase inhibitor vanadate protects osteoblasts from growth inhibition by glucocorticoids.
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Lactoferrin protects osteoblasts and osteocytes from apoptosis
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The expression and regulation of BIM in osteciblasts undergoing growth factor withdrawal
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Glucocorticoids induce rapid up-regulation of mitogen-activated protein kinase phosphatase-1 and dephosphorylation of extracellular signal-regulated kinase and impair proliferation in human and mouse osteoblast cell lines.
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Selective targeting of DR5 circumvents resistance of MG-63 osteosarcoma to trail-induced apoptosis
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Glucocorticoids inhibit osteoblast proliferation via transcriptional upregulation of MKP-1: A prime target for the development of novel osteo-anabolic agents
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Selective targeting of death receptor 5 circumvents resistance of MG-63 osteosarcoma cells to TRAIL-induced apoptosis.
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Human Mesenchymal Stem Cells-Derived Exosome Mimetic Vesicles Regulation of the MAPK Pathway and ROS Levels Inhibits Glucocorticoid-Induced Apoptosis in Osteoblasts
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Osteoblasts protect multiple myeloma cells from T-cell-induced apoptosis
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MECHANISMS OF OSTEOBLAST INHIBITION BY STEROIDS
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Rapid inhibitory effects of glucocorticoids on ERK activity and osteoblast proliferation occur via transcriptional upregulation of MKP-1
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Rapid inhibitory effects of glucocorticoids on ERK activity and osteoblast proliferation occur via transcriptional upregulation of MKP-1
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Glucocorticoid transiently upregulates mitochondrial biogenesis in the osteoblast
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Overexpression of Bcl2 in osteoblasts inhibits osteoblast differentiation and induces osteocyte apoptosis.
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Mitogen-activated protein kinase phosphatase 1/dual specificity phosphatase 1 mediates glucocorticoid inhibition of osteoblast proliferation.
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Glucocorticoids induce senescence in primary human tenocytes: in vitro and in vivo evidence
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Glucocorticoids induce senescence in primary human tenocytes: in vitro and in vivo evidence
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Glucocorticoids induce senescence and alter matrix protein expression in primary human tenocytes
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Glucocorticoids induce senescence and alter matrix protein expression in primary human tenocytes
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REPIN1 regulates iron metabolism and osteoblast apoptosis in osteoporosis
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Protection against glucocorticoid-induced damage in human tenocytes by modulation of ERK, Akt, and forkhead signaling.
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Oxidative stress and glucocorticoids induce forkhead expression and cause degenerative changes in primary human tenocytes
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Protection against glucocorticoid-induced ROS generation and Forkhead-mediated damage response in human tenocytes
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Bone biology and the pathogenesis of osteoporosis.
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Involvement of Bim in Photofrin-Mediated Photodynamically Induced Apoptosis
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Disruption of glucocorticoid signal in osteoblasts and osteocytes attenuates hindlimb unloading-induced bone loss in mice
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Alternative insulin signalling pathways in osteoblasts
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Glucocorticoid-induced osteoporosis in the rat is prevented by the tyrosine phosphatase inhibitor, sodium orthovanadate.
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Allolobophora caliginosa coelomic fluid and extract alleviate glucocorticoid-induced osteoporosis in mice by suppressing oxidative stress and regulating osteoblastic/osteoclastic-related markers
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