Erratum: Association studies of variants in promoter and coding regions of beta-cell ATP sensitive K-channel genes SUR1 and Kir6.2 with Type 2 diabetes mellitus (UKPDS 53) (Diabetic Medicine (2001) 18 (206-212))

Erratum: Association studies of variants in promoter and coding regions of beta-cell ATP sensitive K-channel genes SUR1 and Kir6.2 with Type 2 diabetes mellitus (UKPDS 53) (Diabetic Medicine (2001) 18 (206-212))

Chi tiết về thư mục
Những tác giả chính:	Gloyn, A, Hashim, Y, Ashcroft, S, Ashfield, R, Wiltshire, S, Turner, R
Định dạng:	Journal article
Ngôn ngữ:	English
Được phát hành:	2003

Những quyển sách tương tự

Association studies of variants in promoter and coding regions of beta-cell ATP-sensitive K-channel genes SUR1 and Kir6.2 with Type 2 diabetes mellitus (UKPDS 53).
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Glimepiride block of Kir6.2/SUR1 Kir6.2/SUR2A and Kir6.2/SUR2A and Kir6.2/SUR2B K-ATP channels.
Bằng: Ashcroft, F, et al.
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Nucleotide modulation of pinacidil stimulation of the cloned K(ATP) channel Kir6.2/SUR2A.
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Mapping the architecture of the ATP-binding site of Kir6.2
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Effects of mitiglinide (S 21403) on Kir6.2/SUR1, Kir6.2/SUR2A and Kir6.2/SUR2B types of ATP-sensitive potassium channel.
Bằng: Reimann, F, et al.
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Molecular basis of Kir6.2 mutations causing neonatal diabetes and neonatal diabetes with neurological features
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Time-dependent activation of Kir6.2/SUR2A channels but not Kir6.2/SUR1 channels in the presence of ATP
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Time-dependent activation of KIR6.2/SUR2A channels but not KIR6.2/SUR1 channels in the presence of MgATP
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Identification of residues contributing to the ATP binding site of Kir6.2.
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Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological features.
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Kir6.2 mutations causing neonatal diabetes provide new insights into Kir6.2-SUR1 interactions.
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The role of lysine 185 in the kir6.2 subunit of the ATP-sensitive channel in channel inhibition by ATP.
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Involvement of the N-terminus of Kir6.2 in the inhibition of the KATP channel by ATP.
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Activating mutations in the gene encoding the ATP-sensitive potassium-channel subunit Kir6.2 and permanent neonatal diabetes.
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Mapping the architecture of the ATP-binding site of the KATP channel subunit Kir6.2.
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Focus on Kir6.2: a key component of the ATP-sensitive potassium channel.
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Interaction of the cytosolic domains of the Kir6.2 subunit of the K(ATP) channel is modulated by sulfonylureas.
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Screening for mutations in the Kir6.2 subunit of the beta-cell K-ATP channel
Bằng: Sakura, H, et al.
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Quantitative traits associated with the Type 2 diabetes susceptibility allele in Kir6.2.
Bằng: Weedon, M, et al.
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An in-frame deletion in Kir6.2 (KCNJ11) causing neonatal diabetes reveals a site of interaction between Kir6.2 and SUR1.
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Direct photoaffinity labeling of the Kir6.2 subunit of the ATP-sensitive K+ channel by 8-azido-ATP.
Bằng: Tanabe, K, et al.
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Behavioral phenotyping of mice lacking the K ATP channel subunit Kir6.2.
Bằng: Deacon, R, et al.
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Behavioral phenotyping of mice lacking the K ATP channel subunit Kir6.2.
Bằng: Deacon, R, et al.
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Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptor.
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A novel SUR1/Kir6.2 specific K-ATP channel opener
Bằng: Dabrowski, M, et al.
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A mouse model of neonatal diabetes caused by the K-ATP channel mutation Kir6.2-V59M
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ATP-dependent interaction of the cytosolic domains of Kir6.2 revealed by fluorescence resonance energy transfer
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Permanent neonatal diabetes caused by an in-frame deletion in the N-terminus of Kir6.2
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Kir6.2-dependent high-affinity repaglinide binding to beta-cell K(ATP) channels.
Bằng: Hansen, A, et al.
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Direct photoaffinity labeling of Kir6.2 by [gamma-(32)P]ATP-[gamma]4-azidoanilide.
Bằng: Tanabe, K, et al.
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Mutations within the P-loop of Kir6.2 modulate the intraburst kinetics of the ATP-sensitive potassium channel.
Bằng: Proks, P, et al.
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Functional analysis of a structural model of the ATP-binding site of the KATP channel Kir6.2 subunit.
Bằng: Antcliff, J, et al.
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Neonatal diabetes - The role of KCNJ11 (Kir6.2)
Bằng: Tammaro, P
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A mutation in the ATP-binding site of the Kir6.2 subunit of the KATP channel alters coupling with the SUR2A subunit.
Bằng: Tammaro, P, et al.
Được phát hành: (2007)

Genetics: how the UKPDS contributed to determining the genetic landscape of Type 2 diabetes.
Bằng: Gloyn, A, et al.
Được phát hành: (2008)

Phentolamine block of KATP channels is mediated by Kir6.2.
Bằng: Proks, P, et al.
Được phát hành: (1997)

Functional analysis of six Kir6.2 (KCNJ11) mutations causing neonatal diabetes.
Bằng: Girard, C, et al.
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The Kir6.2-F333I mutation differentially modulates K-ATP channels composed of SUR1 or SUR2 subunits
Bằng: Tammaro, P, et al.
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The Kir6.2-F333I mutation differentially modulates K ATP channels composed of SUR1 and SUR2 subunits
Bằng: Tammaro, P, et al.
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Adjacent mutations in the gating loop of Kir6.2 produce neonatal diabetes and hyperinsulinism.
Bằng: Shimomura, K, et al.
Được phát hành: (2009)