The exon A (C77G) mutation is a common cause of abnormal CD45 splicing in humans

The exon A (C77G) mutation is a common cause of abnormal CD45 splicing in humans

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Bibliografske podrobnosti
Main Authors:	Tchilian, E, Wallace, D, Imami, N, Liao, H, Burton, C, Gotch, F, Martinson, J, Haynes, B, Beverley, P
Format:	Journal article
Izdano:	2001

Podobne knjige/članki

The exon A (C77G) mutation is a common cause of abnormal CD45 splicing in humans.
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A point mutation in CD45 may be associated with an increased risk of HIV-1 infection
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Novel perforin mutation in a patient with hemophagocytic lymphohistiocytosis and CD45 abnormal splicing.
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A point mutation within CD45 exon A is the cause of variant CD45RA splicing in humans.
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A CD45 polymorphism associated with abnormal splicing is absent in African populations.
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A CD45 polymorphism associated with abnormal splicing is absent in African populations
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A point mutation in CD45 may be associated with an increased risk of HIV-1 infection (vol 15, pg 1892, 2001)
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Erratum: A point mutation in CD45 may be associated with an increased risk of HIV-1 infection (AIDS (2001) 15 (1892-1894)). [2]
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Abnormal cell surface antigen expression in individuals with variant CD45 splicing and histiocytosis.
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Unusual case presentations associated with the CD45 C77G polymorphism
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Unusual case presentations associated with the CD45 C77G polymorphism.
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CD45 in memory and disease.
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CD45 in memory and disease
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Altered CD45 expression and disease.
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CD45 variant alleles: possibly increased frequency of a novel exon 4 CD45 polymorphism in HIV seropositive Ugandans.
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Analysis of the C77G polymorphism in the CD45 gene in patients affected by Langerhans cell histiocytosis
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Altered CD45 expression in C77G carriers influences immune function and outcome of hepatitis C infection.
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Altered CD45 expression in C77G carriers influences immune function and outcome of hepatitis C infection
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Regulation of alternative splicing of CD45 by antagonistic effects of SR protein splicing factors.
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Geographical distribution and disease associations of the CD45 exon 6 138G variant.
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Hotspot exons are common targets of splicing perturbations
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CD45 regulates apoptosis in peripheral T lymphocytes
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CD45 regulates apoptosis in peripheral T lymphocytes.
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A high-frequency polymorphism in exon 6 of the CD45 tyrosine phosphatase gene (PTPRC) resulting in altered isoform expression.
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Phenotypic changes associated with activation of CD45RA+ and CD45RO+ T cells.
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A deletion in the gene encoding the CD45 antigen in a patient with SCID.
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A deletion in the gene encoding the CD45 antigen in a patient with SCID
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Altered CD45 isoform expression affects lymphocyte function in CD45 Tg mice.
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CD45 negatively regulates tumour necrosis factor and interleukin-6 production in dendritic cells
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CD45 negatively regulates tumour necrosis factor and interleukin-6 production in dendritic cells.
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Combinations of CD45 isoforms are crucial for immune function and disease
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Combinations of CD45 isoforms are crucial for immune function and disease.
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A putative exonic splicing enhancer in exon 7 of the PDHA1 gene affects splicing of adjacent exons.
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Regulation of CD44 binding to hyaluronan by glycosylation of variably spliced exons.
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Categorization of 77 <it>dystrophin </it>exons into 5 groups by a decision tree using indexes of splicing regulatory factors as decision markers
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CD45 and the thymus.
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