CD44 Blockade Generates Functional iTreg In Vitro By Reducing Memory T Cell IL-6 Production

CD44 Blockade Generates Functional iTreg In Vitro By Reducing Memory T Cell IL-6 Production

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Bibliografiske detaljer
Main Authors:	Whatcott, A, Wood, K, Bushell, A
Format:	Conference item
Udgivet:	2014

Lignende værker

CD44 Blockade Generates Functional iTreg In Vitro By Reducing Memory T Cell IL-6 Production.
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Udgivet: (2014)

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Cholesterol suppresses human iTreg differentiation and nTreg function through mitochondria-related mechanisms
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TIGIT+ iTregs elicited by human regulatory macrophages control T cell immunity
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PRMT5 regulates epigenetic changes in suppressive Th1-like iTregs in response to IL-12 treatment
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Vitamin C stabilizes CD8+ iTregs and enhances their therapeutic potential in controlling murine GVHD and leukemia relapse
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The small molecule inhibitor BX-795 uncouples IL-2 production from inhibition of Th2 inflammation and induces CD4+ T cells resembling iTreg
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Sustained store-operated calcium entry utilizing activated chromatin state leads to instability in iTregs
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ACLY ubiquitination by CUL3-KLHL25 induces the reprogramming of fatty acid metabolism to facilitate iTreg differentiation
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Ash1l and lnc-Smad3 coordinate Smad3 locus accessibility to modulate iTreg polarization and T cell autoimmunity
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iTreg cells-secreted IL10 alleviates lupus nephritis through inactivating lncRNA HAR1A transcription to suppress SMARCD1-mediated iNOS activation
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254 CTLA-4 blockade promotes Treg glucose metabolism and reduces Treg functional stability in glycolysis-defective tumors
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Evaluation of CD44s, CD44v6, CXCR2, CXCL1, and IL-1β in Benign and Malignant Tumors of Salivary Glands
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CD44s和 CD44v6 在肺癌中的表达
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Blockade of the pentraxin 3/CD44 interaction attenuates lung injury‐induced fibrosis
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Tolerogenic vaccination reduced effector memory CD4 T cells and induced effector memory Treg cells for type I diabetes treatment.
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CD8+CD122+PD-1+ Tregs Synergize With Costimulatory Blockade of CD40/CD154, but Not B7/CD28, to Prolong Murine Allograft Survival
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Altered Treg and IL-1A Expression in the Immune Microenvironment  of Lung Squamous-cell Cancer after EGFR Blockade
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TNFR2 blockade promotes antitumoral immune response in PDAC by targeting activated Treg and reducing T cell exhaustion
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